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Titlebook: Mitochondrial DNA Mutations in Aging, Disease and Cancer; Keshav K. Singh Book 1998 Springer-Verlag Berlin Heidelberg 1998 Alzheimer Krank

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書(shū)目名稱(chēng)Mitochondrial DNA Mutations in Aging, Disease and Cancer
編輯Keshav K. Singh
視頻videohttp://file.papertrans.cn/636/635052/635052.mp4
概述Most comprehensive, up-to-date analysis of the role of mitochondria in aging and diseases.
圖書(shū)封面Titlebook: Mitochondrial DNA Mutations in Aging, Disease and Cancer;  Keshav K. Singh Book 1998 Springer-Verlag Berlin Heidelberg 1998 Alzheimer Krank
描述Many human genetic diseases associated with blood, brain, colon, ear, eye, heart, kidney, liver, muscle, and pancreas are caused by mutations in mitochondrial DNA. Mutations in DNA can result in defects of the electron transport complexes, intermediates of the tricarboxylic acid cycle and substrate transport. The clinical manifestation of these diseases often involves muscle and the nervous system. Mitochondrial DNA mutations have now been associated with aging as well as age-related degenerative diseases such as Parkinson‘s, Alzheimer‘s, and Huntington‘s diseases. Changes in structure, function, and a number of mitochondria play an important role in carcinogenesis. Furthermore, the role of mitochondria in the execution of programmed cell death or apoptosis has been recognized recently.
出版日期Book 1998
關(guān)鍵詞Alzheimer Krankheit; Alzheimer‘s disease; DNA; Huntington‘s diesease; Huntington-Krankheit; Mitochondrien
版次1
doihttps://doi.org/10.1007/978-3-662-12509-0
isbn_softcover978-3-662-12511-3
isbn_ebook978-3-662-12509-0
copyrightSpringer-Verlag Berlin Heidelberg 1998
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Book 1998ated with aging as well as age-related degenerative diseases such as Parkinson‘s, Alzheimer‘s, and Huntington‘s diseases. Changes in structure, function, and a number of mitochondria play an important role in carcinogenesis. Furthermore, the role of mitochondria in the execution of programmed cell death or apoptosis has been recognized recently.
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Mitochondrial DNA Mutations in Aging, Disease and Cancer
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https://doi.org/10.1007/978-3-662-12509-0Alzheimer Krankheit; Alzheimer‘s disease; DNA; Huntington‘s diesease; Huntington-Krankheit; Mitochondrien
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