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Titlebook: Microbial Endocrinology: The Microbiota-Gut-Brain Axis in Health and Disease; Mark Lyte,John F. Cryan Book 2014 Springer New York 2014 dis

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發(fā)表于 2025-3-21 19:56:31 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Microbial Endocrinology: The Microbiota-Gut-Brain Axis in Health and Disease
編輯Mark Lyte,John F. Cryan
視頻videohttp://file.papertrans.cn/633/632948/632948.mp4
概述Provides readers with a full understanding of the capabilities of the microbiota to interface with the brain and form the microbiota-gut-brain axis and a detailed examination of the consequences of th
叢書(shū)名稱(chēng)Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Microbial Endocrinology: The Microbiota-Gut-Brain Axis in Health and Disease;  Mark Lyte,John F. Cryan Book 2014 Springer New York 2014 dis
描述.The field of microbial endocrinology is expressly devoted to understanding the mechanisms by which the microbiota (bacteria within the microbiome) interact with the host (“us”).?This interaction is a two-way street and the driving force that governs these interactions are the neuroendocrine products of both the host and the microbiota. Chapters include neuroendocrine hormone-induced changes in gene expression and microbial endocrinology and probiotics. This is the first in a series of books?dedicated to understanding how bi-directional communication between host and bacteria represents the cutting edge of translational medical research, and hopefully identifies new ways to understand the mechanisms that determine health and disease.?.
出版日期Book 2014
關(guān)鍵詞disease; microbial endocrinology; microbiome; physiology
版次1
doihttps://doi.org/10.1007/978-1-4939-0897-4
isbn_softcover978-1-4939-5501-5
isbn_ebook978-1-4939-0897-4Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightSpringer New York 2014
The information of publication is updating

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發(fā)表于 2025-3-21 23:12:55 | 只看該作者
Book 2014teract with the host (“us”).?This interaction is a two-way street and the driving force that governs these interactions are the neuroendocrine products of both the host and the microbiota. Chapters include neuroendocrine hormone-induced changes in gene expression and microbial endocrinology and prob
板凳
發(fā)表于 2025-3-22 03:59:59 | 只看該作者
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發(fā)表于 2025-3-22 06:44:43 | 只看該作者
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發(fā)表于 2025-3-22 11:07:09 | 只看該作者
Microbial Endocrinology and the Microbiota-Gut-Brain Axishost behavior, microbial endocrinology with its theoretical basis rooted in shared neuroendocrine signaling mechanisms provides for testable experiments with which to understand the role of the microbiota in host behavior and as importantly the ability of the host to influence the microbiota through neuroendocrine-based mechanisms.
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發(fā)表于 2025-3-22 14:08:51 | 只看該作者
Utilizing “Omics” Tools to Study the Complex Gut Ecosystemeview, we consider how new “omics” technology can be applied to the study of the gut ecosystem in human and animal models with special consideration given to factors that should be considered in the design of experiments and clinical trials.
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發(fā)表于 2025-3-22 18:51:02 | 只看該作者
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發(fā)表于 2025-3-22 22:32:36 | 只看該作者
0065-2598 in axis and a detailed examination of the consequences of th.The field of microbial endocrinology is expressly devoted to understanding the mechanisms by which the microbiota (bacteria within the microbiome) interact with the host (“us”).?This interaction is a two-way street and the driving force th
9#
發(fā)表于 2025-3-23 02:15:29 | 只看該作者
Microbial Endocrinology and the Microbiota-Gut-Brain Axisthin the microorganisms themselves or within the host they inhabit. As such, microbial endocrinology represents the intersection of the fields of microbiology and neurobiology. The acquisition of neurochemical-based cell-to-cell signaling mechanisms in eukaryotic organisms is believed to have been a
10#
發(fā)表于 2025-3-23 06:33:28 | 只看該作者
Utilizing “Omics” Tools to Study the Complex Gut Ecosystem presence of pathogens. When the balance between host and microbes is disrupted, risk for disease increases. There is mounting evidence that microbial dysbiosis is a substantial risk factor for common gut diseases including IBS, IBD and colorectal cancer. Understanding this dysbiosis is challenging
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