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Titlebook: Metabolism and Epigenetic Regulation: Implications in Cancer; Tapas Kumar Kundu,Chandrima Das Book 2022 The Editor(s) (if applicable) and

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21#
發(fā)表于 2025-3-25 03:34:26 | 只看該作者
22#
發(fā)表于 2025-3-25 09:47:07 | 只看該作者
Sweta Sikder,Atanu Mondal,Chandrima Das,Tapas K. Kundu
23#
發(fā)表于 2025-3-25 15:20:00 | 只看該作者
24#
發(fā)表于 2025-3-25 16:02:47 | 只看該作者
Payel Mondal,Niharika Tiwary,Amrita Sengupta,Sinjini Dhang,Siddhartha Roy,Chandrima Das
25#
發(fā)表于 2025-3-25 23:33:04 | 只看該作者
26#
發(fā)表于 2025-3-26 00:54:49 | 只看該作者
Deepak Pant,Srinivas Abhishek Mutnuru,Sanjeev Shukla
27#
發(fā)表于 2025-3-26 07:49:28 | 只看該作者
Reprogramming Carbohydrate Metabolism in Cancer and Its Role in Regulating the Tumor Microenvironmenluding amino acid and lipid metabolism and pentose phosphate pathway are diverged. In contrast to normal cells, increased glycolytic flux is a distinguishing feature of the highly proliferative cancer cells, which supports them to adapt to a hypoxic environment and also protects them from oxidative
28#
發(fā)表于 2025-3-26 10:28:46 | 只看該作者
Role of the Histone Acetyl Transferase MOF and the Histone Deacetylase Sirtuins in Regulation of H4K(males absent on the first) and the histone deacetylases (HDACs) Sirtuins on histone H4K16 acetylation (H4K16ac)?and DNA damage repair. We also discuss the role of this important histone modification in light of metabolic rewiring and its role in regulating human pathophysiologic states.
29#
發(fā)表于 2025-3-26 14:38:10 | 只看該作者
Nutritional Epigenetics: How Metabolism Epigenetically Controls Cellular Physiology, Gene Expressionpigenetic profile. Such long-term or repeated alteration of epigenetic profile can lead to several diseases, like cancer, insulin resistance and diabetes, cognitive impairment, neurodegenerative disease, and metabolic syndromes. Here we discuss the functions of key nutrients that contribute to epige
30#
發(fā)表于 2025-3-26 19:02:56 | 只看該作者
Epigenetic Reprogramming of the Glucose Metabolic Pathways by the Chromatin Effectors During Cancerrole of metabolic intermediates in the cell. Also, we summarize the influence of histone effectors in reprogramming the cancer cell metabolism. An interesting aspect of this chapter includes the detailed methods to detect the aberrant metabolic flux, which can be instrumental for the therapeutic reg
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