Titlebook: Metabolic Interaction in Infection; Ricardo Silvestre,Egídio Torrado Book 2018 Springer International Publishing AG, part of Springer Natu

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Interplay Between Metabolic Sensors and Immune Cell Signalinge in the form of infection, tumors, autoantigens, and can occur after mismatched transplantation. During response, immune cells alter their metabolic activities. However, the subsets of the same cell type differ to distinctively associate specific immune function to a particular metabolic profile. T

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Alterations on Cellular Redox States upon Infection and Implications for Host Cell Homeostasisbalance between the oxidized and reduced forms of these molecules is tightly regulated by intracellular metabolism assuring the maintenance of homeostatic conditions, which are essential for cell survival and proliferation. A recent cluster of data has highlighted the importance of the intracellular

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Metabolomic-Based Methods in Diagnosis and Monitoring Infection Progressionapter, a general workflow for metabolomics is summarized. Subsequently, an overview of the major contributions of this omics science to the field of biomarkers of infectious diseases is discussed. Different approaches using a variety of analytical platforms can be distinguished to unveil the key met

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Metabolic Host Response to Intracellular Infectionslearance to the establishment of infection. At its core, these interactions result in multiple metabolic adaptations of both the pathogen and its host cell. There is growing evidence that the host metabolic response plays a key role in the development of immune responses against the invading pathoge

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Metabolic Regulation of Innate Immunity to Fungal Infectionrocesses during innate and adaptive immune responses. Although the specific requirements of myeloid cells after the canonical lipopolysaccharide/TLR4 stimulation have been extensively addressed, recent evidence suggests that this model may not represent a universally accurate metabolic blueprint. In

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Metabolic Crosstalk Between Host and Parasitic Pathogenstritional pressures exerted by both elements of this duet thus dictate this host–parasite niche. To survive and proliferate inside a host and a harsh nutritional environment, the parasites modulate different nutrient sensing pathways to subvert host metabolic pathways. Such mechanism is able to chan