Titlebook: Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma; Alexander Baethmann,Oliver S. Kempski,Frank Staub Conference proceed

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書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma影響因子(影響力)




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma影響因子(影響力)學(xué)科排名




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma網(wǎng)絡(luò)公開度




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma被引頻次




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma被引頻次學(xué)科排名




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma年度引用




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma年度引用學(xué)科排名




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma讀者反饋




書目名稱Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma讀者反饋學(xué)科排名

斷言

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KIN

OTTER

Significance of the Inflammatory Response in Brain Ischemiaat leukocytes accumulate in focal zones of acute brain ischemia at a sufficiently early stage to participate in the process of progressive ischemic brain damage and that partial inhibition of that accumulation, by various measures, can attenuate ischemic brain injury. Mechanisms of leukocyte adhesio

現(xiàn)暈光

NEG

Aboveboard

Three-Dimensional Metabolic and Hemodynamic Imaging of the Normal and Ischemic Rat Brainlopment of powerful image-processing techniques permitting three-dimensional (3D) autoradiographic image-averaging and analysis of replicate studies by a novel method termed “disparity analysis”. This method, based upon a linear affine transformation model, directly estimates scaling, translation an

neuron

Origins of Glutamate Release in Ischaemiashown that the increase in extracellular glutamate under global penumbral conditions is minor. However, in the border of the ischaemic core, recurrent spreading depression is presumably associated with transient vesicular release of glutamate (exocytosis). With ischaemic insults severe enough to pro

自制

Swelling, Intracellular Acidosis, and Damage of Glial Cellsrobic glycolysis. Resulting metabolites, such as arachidonic- and lactic acid, can be expected to leak into perifocal brain areas, contributing there to cytotoxic swelling and damage of neurons and glia. Since elucidation of mechanisms underlying cell swelling and damage in the brain is difficult .,