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Titlebook: Mechanisms of Secondary Brain Damage from Trauma and Ischemia; Recent Advances of o A. Baethmann,J. Eriskat,N. Plesnila Conference proceedi

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書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia
副標題Recent Advances of o
編輯A. Baethmann,J. Eriskat,N. Plesnila
視頻videohttp://file.papertrans.cn/629/628765/628765.mp4
概述Top level clinical and laboratory scientists present updates and exchanges of their most recent clinical and experimental findings and concepts.Includes supplementary material:
叢書名稱Acta Neurochirurgica Supplement
圖書封面Titlebook: Mechanisms of Secondary Brain Damage from Trauma and Ischemia; Recent Advances of o A. Baethmann,J. Eriskat,N. Plesnila Conference proceedi
出版日期Conference proceedings 2004
關(guān)鍵詞brain; brain injury; cerebral ischemia; electroencephalography (EEG); head; head injury; pathophysiology; r
版次1
doihttps://doi.org/10.1007/978-3-7091-0603-7
isbn_softcover978-3-7091-7206-3
isbn_ebook978-3-7091-0603-7Series ISSN 0065-1419 Series E-ISSN 2197-8395
issn_series 0065-1419
copyrightSpringer-Verlag Wien 2004
The information of publication is updating

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Lessons from epidemiologic studies in clinical trials of traumatic brain injuryTirilizad Trials which enrolled over 1,100 patients. A major observation from that trial was that pretreatment hypotension or hypoxia could be unbalanced even in a large two arm blinded study. Another issue of the tirilazad trial was the influence of gender affecting not only outcome but also drug metabolism.
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Pharmacological preconditioning in global cerebral ischemia function. Bcl-2-protein is expressed in neurons that survive cerebral ischemia and may parallel the time course of tolerance after ischemic preconditioning. With this study we examined whether differences in bcl-2-protein expression compared to baseline may be involved in the induction of ischemic tolerance using 3-NPA.
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