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Titlebook: Mechanisms of Plant Defense Responses; Bernard Fritig,Michel Legrand Book 1993 Springer Science+Business Media Dordrecht 1993 Expression.P

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發(fā)表于 2025-3-21 18:51:40 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Mechanisms of Plant Defense Responses
編輯Bernard Fritig,Michel Legrand
視頻videohttp://file.papertrans.cn/629/628753/628753.mp4
叢書名稱Developments in Plant Pathology
圖書封面Titlebook: Mechanisms of Plant Defense Responses;  Bernard Fritig,Michel Legrand Book 1993 Springer Science+Business Media Dordrecht 1993 Expression.P
描述Research on the mechanisms of plant defense responses to stress and pathogen attack has attracted much attention in recent years. This increasing interest stems from the fact that the tools of molecular biology now enable us to study the molecular basis of old biological concepts such as host-pathogen recognition (and particularly the gene- for-gene relationship), hypersensitive cell death and systemic acquired resistance. Our knowledge about avirulence and resistance genes, elicitors, signal transduction and genes involved in plant defense is rapidly expanding. Moreover we are just beginning to test in planta the potential of these results for biotechnological applications, aimed at improving plant resistance to diseases. The 2nd Conference of the European Foundation for Plant Pathology, hosted by the "Societe Fran~aise de Phytopathologie", was devoted to "Mechanisms of plant defense responses" and was held in Strasbourg, France. It brought together over 350 scientists from universities, research institutes and private sectors of 24 countries. Major advances in the areas under study have been reviewed in plenary lectures and are developed in the main articles of this book. Over 16
出版日期Book 1993
關(guān)鍵詞Expression; Pathogen; biotechnology; gene expression; regulation
版次1
doihttps://doi.org/10.1007/978-94-011-1737-1
isbn_softcover978-94-010-4761-6
isbn_ebook978-94-011-1737-1Series ISSN 0929-1318
issn_series 0929-1318
copyrightSpringer Science+Business Media Dordrecht 1993
The information of publication is updating

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A Protein from Apple Leaves Inhibits Pectinolytic Activity of ,oung, scab-susceptible leaves of “Golden Delicious” (GD) contained significantly (p < 0.05) more PGIP than old, scab-resistant GD-leaves. We concluded that there is no simple positive correlation between the resistance of an apple leaf against . and the PGIP-content of the leaf.
板凳
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Book 1993rest stems from the fact that the tools of molecular biology now enable us to study the molecular basis of old biological concepts such as host-pathogen recognition (and particularly the gene- for-gene relationship), hypersensitive cell death and systemic acquired resistance. Our knowledge about avi
地板
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Repetitive Motifs in the , Avirulence Gene Family Determine Specificity of Resistance to , pv. , activity on tomato. The AvrBs3-2 protein is 97% identical to AvrBs3. The difference in specificity is most likely due to the different order of the repeats in the Avr-protein. Interestingly, C-terminal deletion derivatives of . are still active on tomato.
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Purification and Characterisation of Pectin Lyase from , Subsp. , Strain Scri 10434 × 10. mol min. ml. respectively. The presence of divalent cations, although not essential, enhanced pectin lyase activity. It was also shown that, ., strain SCRI 1043 produces only one isomer of pectin lyase.
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Functional Analysis of the , Resistance Locus in Barleyon process combined with gene dosage experiments provides strong evidence that hypersensitive cell death is causally not required tor . mediated arrest of fungal growth. We speculate that gene dosage experiments may provide a means to separate primary from secondary defense reactions.
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Genetic Approaches to an Understanding of Specific Resistance Responses of , Against Phytopathogenictaining the resistant state. Finally, we hope to integrate this knowledge with definition of specific . genes, and the genes encoding passage of . signals through the bacterial membrane. This presentation outlined our current progress in these areas.
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