Titlebook: Mechanisms and Models in Toxicology; Proceedings of the E Claire M. Chambers,Philip L. Chambers,Donald S. Da Conference proceedings 1987 Sp

infelicitous

https://doi.org/10.1007/978-3-642-72558-6biotechnology; cancer; cancer risk; toxicity; toxicology

拋射物

Persistent Organ Damage and Cancer Production in Rats and MiceThe reparative processes that take place in response to tissue injury are indubitably of great importance in restoring the integrity of a damaged organ, but they are not usually thought of as a mechanism.

粗俗人

圓錐

Apoptosis: Cell Death Under Homeostatic Controle reviewed which shows that cells can (and in most cases eventually do) die in response to physiological stimuli. Physiological cell death will be seen to be mediated by cer-tain intracellular events, which can be defined in molecular terms, and which are themselves internally regulated.

Hallowed

羽毛長(zhǎng)成

四溢

Apoptosis: Cell Death Under Homeostatic Controle reviewed which shows that cells can (and in most cases eventually do) die in response to physiological stimuli. Physiological cell death will be seen to be mediated by cer-tain intracellular events, which can be defined in molecular terms, and which are themselves internally regulated.

indicate

On The Role of Calcium in Chemical Toxicityn the organ-ism. It is equally clear that there exist protective systems which can trap or inac-tivate toxic metabolites and thereby prevent their accumulation within the tissues and subsequent toxicity. Although phase I reactions, in particular those mediated by the cytochrome P.-linked monooxygena

受辱

follicular-unit

Some Aspects of Cell Defense Mechanisms of Glutathione and Vitamin E During Cell Injury molecular oxygen (Reed 1985). Less well understood is the possible interaction of glutathione and vitamin E in chemically induced cell injury, as well as the oxidation type of stress caused by incubation of cells in calcium-depleted medium. This laboratory has demonstrated that chemically induced i