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Titlebook: Maturation Phenomenon in Cerebral Ischemia V; Fifth International Alastair M. Buchan,Umeo Ito,Igor Klatzo Conference proceedings 2004 Spri

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樓主: cobble
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發(fā)表于 2025-3-23 12:45:51 | 只看該作者
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發(fā)表于 2025-3-23 16:13:17 | 只看該作者
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發(fā)表于 2025-3-23 18:57:27 | 只看該作者
Compartmentalization in Focal Cerebral Ischemiacular compartment into the extravascular compartment, and disturbance of cell-cell relationships within the extravascular compartment, proteases identified in the target tissue following the ischemic insult which lesion major alterations in tissue barriers occur. These involve disruption of the cere
14#
發(fā)表于 2025-3-24 01:31:39 | 只看該作者
Modulation of the Post-Ischemic Immune Response Improves Outcome in Focal Cerebral Ischemia: A Role he blood-brain barrier (BBB) exposes the brain to the peripheral circulation and thus the systemic immune system. Following stroke, antigens found either primarily or exclusively in the central nervous system (CNS), like neuron-specific enolase (NSE), glial fibrillary acidic protein (GFAP), myelin b
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發(fā)表于 2025-3-24 04:40:44 | 只看該作者
Tumor Necrosis Factor-Alpha (TNF-a) and Ceramide Induce Tolerance to Ischemic and Hypoxic Insults toning with these signaling molecules alters the function of NF.B. Although the nuclear translocation and DNA binding of the NF.B heterodimer, p50 and p65/RelA, are unaffected, p65 binding to the coactivator, p300/CBP, becomes disrupted.
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發(fā)表于 2025-3-24 06:41:42 | 只看該作者
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發(fā)表于 2025-3-24 10:51:00 | 只看該作者
A Role for Cerebrovascular Endothelium in Ischemia and Reperfusionated with various metabolic pathways that are essential to sustain the dynamic homeostasis of the brain [., .]. Many of these processes are thought to be neuronally regulated [.]. It is now widely accepted that cerebral vascular endothelial cells not only constitute a permeability barrier to ions an
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發(fā)表于 2025-3-24 15:13:14 | 只看該作者
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發(fā)表于 2025-3-24 22:02:41 | 只看該作者
Acid-Sensitive Ion Channels in Brain: New Modulation of Ischemic Injuryepithelial sodium channels (ENaCs) in mammals, the stretch activated degenerins (Mec-4, -10; Unc-105)in.and the ligand-gated FMRF-amide channel in.All are inhibited by amiloride, are permeable to Na+, and share a common structure consisting of short intracellular amino-and carboxy-terminal domains c
20#
發(fā)表于 2025-3-24 23:36:52 | 只看該作者
igned to ensure ease of reference for clinicians.This text presents a compilation of topics that have been taught at Metabolic University (MU), an interactive, didactic educational program that has trained over 600 metabolic dietitians/nutritionists, physicians, nurses and genetic counselors...This
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