Titlebook: Maturation Phenomenon in Cerebral Ischemia II; Neuronal Recovery an Umeo Ito,Takaaki Kirino,Igor Klatzo (Former Chief Conference proceedin

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T. Kogure,Y. Yamasaki,Y. Matsuo,K. Kogureod pressure, ., hypertension, increases the risk of ischemic heart disease, stroke, peripheral vascular disease and other CVDs. .. The low intake of fruit and vegetables and whole grain fiber and high consumption of saturated fat and high-cholesterol diets can lead to hypercholesterolemia and athero

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Molecular Approaches to Neuronal Functions and Neuronal Cell Deathis hoped that some of them may contribute to a molecular understanding of neuronal functions and neuronal cell death, and also to the development of new clinical methods for rescuing brain damage lesions..Some of our data on molecular and cellular biology of neuronal functions and neuronal cell deat

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Ischemic Neuronal Injury and Gene Expression of Facilitative and Inhibitory Growth Factorsitory factor (GIF) in ischemic brain tissue. The mRNA expression of these growth factors was studied using the in situ hybridization method and compared with the expression of c-., c-., and heat shock protein(HSP) 70 mRNA in the serial sections of the brain tissue during the recirculation period aft

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Is Apoptosis Involved in Glutamate Neurotoxicity? the possible involvement of apoptosis in such death..In the first part of the study we employed Nomarski optics combined with a digital image processor to examine the acute type of glutamate neurotoxicity, and found a rapid change in the nucleus followed by cellular swelling. These changes could be

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Glutamate Enhances Brain Damage from Ischemia and Traumaemia or other adverse conditions. Intensive research of the role of mediator compounds is clinically worthwile as more specific forms of treatment may emerge for the benefit of afflicted patients. In view of the great number of factors, cytokines, etc., which could play a role, it is mandatory that

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