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Titlebook: Mathematical Modelling and Simulation of Electrical Circuits and Semiconductor Devices; Proceedings of a Con R. E. Bank,K. Merten,R. Bulirs

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41#
發(fā)表于 2025-3-28 14:53:26 | 只看該作者
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發(fā)表于 2025-3-28 19:07:27 | 只看該作者
43#
發(fā)表于 2025-3-29 01:15:44 | 只看該作者
44#
發(fā)表于 2025-3-29 05:03:27 | 只看該作者
Uri Aschernces amongst workers specializingin different wavebands as well as discussing how multibandobservations can reveal fundamental relationships in our understandingof the Universe.978-0-7923-4803-0978-94-009-1485-8Series ISSN 1743-9213 Series E-ISSN 2352-2186
45#
發(fā)表于 2025-3-29 11:17:13 | 只看該作者
46#
發(fā)表于 2025-3-29 14:00:00 | 只看該作者
Rainer Sawatzkiacterised patients with allergic bronchopulmonary aspergillosis (ABPA), allergic asthmatics with or without . sensitisation, and healthy control persons. The clinical parameters used for the diagnosis of allergy to . were RAST and skin test reactivity to commercial . extracts. IgE antibodies recogni
47#
發(fā)表于 2025-3-29 18:18:01 | 只看該作者
ced response (the improvement of the ventricular relaxation) is related to the severity of the impairment of relaxation as well as the amplitude of the vibration applied. However, it is still unknown whether diastolic vibration increases the coronary flow rate in the heart with impaired relaxation.
48#
發(fā)表于 2025-3-29 21:03:51 | 只看該作者
s proved to be relatively constant under various physiological conditions but have been shown to be altered under pathophysiological conditions such as myocardial stunning and acidosis. In the stunned and acidotic hearts, the 0. costs of Emax were higher than in the normal hearts without marked chan
49#
發(fā)表于 2025-3-30 01:31:59 | 只看該作者
K. Mertencardiomyopathies. Plasma concentrations of interleukin ia (IL-1α), interleukin 1β (IL-1β), and tumor necrosis factor α (TNF-α) were increased in patients with acute myocarditis. TNF-α was also increased in those with dilated cardiomyopathy and hypertrophic cardiomyopathy. These results suggest that
50#
發(fā)表于 2025-3-30 05:32:02 | 只看該作者
Eberhard Griepentroging that LVH is not a simple adaptation to an increased external load. Many investigators have extensively studied the activation of a novel program of gene expression in the heart induced by pressure overload (i.e., the transient induction of immediate early genes and the reexpression of fetal-type
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