Titlebook: Mammary Tumorigenesis and Malignant Progression; Advances in Cellular Robert B. Dickson,Marc E. Lippman Book 1994 Springer Science+Business

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書目名稱Mammary Tumorigenesis and Malignant Progression影響因子(影響力)




書目名稱Mammary Tumorigenesis and Malignant Progression影響因子(影響力)學(xué)科排名




書目名稱Mammary Tumorigenesis and Malignant Progression網(wǎng)絡(luò)公開度




書目名稱Mammary Tumorigenesis and Malignant Progression網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Mammary Tumorigenesis and Malignant Progression被引頻次




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Genetic analysis of breast and ovarian cancer in families.] and of ovarian cancer [.] can be attributed to inheritance of a gene conferring high risk, followed by genetic changes specific to the target epithelial cells of the breast or ovary. Given its very high incidence, breast cancer may also appear more than once in a family purely by bad luck, not be

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含糊其辭

Tumor-suppressor genes in breast cancer progressionreast cancer, modifications of this model are necessary to account for differences in malignant progression between colon and breast cancers. Unlike colon cancer, there is much evidence suggesting that breast cancer is many different diseases. Supporting this hypothesis are the observations that mol

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Estrogen receptor variants in breast cancerreported that patients with inoperable breast tumors frequently responded to surgical castration [.], still a first-line therapeutic modality in premenopausal patients. Other manipulations designed to lower the concentrations of circulating E2 or to block its effects are also as effective, inducing

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晚間

The epidermal growth factor family in the mammary gland and other target organs for ovarian steroidsthe research investigator, and has emphasized the need to understand the pathways by which these compounds exert their effects on target organs. The clinical significance of the ovaries to breast cancer was evident around the turn of the century, when ovariectomy was shown to ameliorate the course o

Semblance

Molecular and clinical aspects of the Neu/ErbB-2 receptor tyrosine kinase,.]. Tumor development in humans is thought to reflect the multiplicity of events in the accumulation of independent mutations that affect clonal growth of the cancerous cell [.]. The identity of the group of genes that confer malignancy in vitro appears to be similar to the set of genes that are mu

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