Titlebook: Malignant Lymphomas and Hodgkin’s Disease: Experimental and Therapeutic Advances; Proceedings of the S Franco Cavalli,G. Bonadonna,Marcel R

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Schlemms-Canal

Origin and biologic function of Reed-Sternberg cells response, as manifested . by impaired delayed hypersensitivity skin tests, and . by decreased E rosette formation as well as decreased T-cell proliferation in response to mitogens or antigens. Abnormalities of cellular immunity were always more pronounced in patients with advanced stages of disease

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Non-Hodgkin’s lymphoma with multilobated nuclei is not a distinct pathologic entityd in extranodal sites and were associated with a favorable clinical course. Consecutively T-lymphocyte derived tumors with multilobated cells have been reported to occur in lymph nodes [2–5] and in the skin [6, 7]. A separate entity may comprise human T-cell leukemia virus (HTLV) related adult T-cel

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Burkitt’s lymphoma: Multiparametric analysis of 55 cell lines with special reference to morphometryy from other related B-cell lymphomas, especially the small non-cleaved or undifferentiated non-Burkitt lymphomas, remain a matter of debate [2–4]. Morphologic differences between the two types of small non-cleaved lymphoma, Burkitt and non-Burkitt, as defined by the ‘Working Formulation for Clinica

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Banded chromosome abnormalities in non-Hodgkin’s lymphoma. Correlations with morphology, immunologicomes have generally been based on small series (less than 50 cases) and have often included many incomplete karyotypes. Bone marrow, blood or effusions have frequently been studied rather than lymph nodes or other primary tumor masses, and relatively few patients have been studied at diagnosis. More

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Chromosomal aberrations in leukemic low-grade malignant B-cell lymphoproliferative neoplasiasis usually called chronic lymphocytic leukemia (CLL). When it became obvious that CLL was a heterogenous disease, it was subdivided in several groups, e.g. according to the Kiel classification [1]. New entities such as immunocytoma, prolymphocytic leukemia, and centrocytic lymphoma, were separated f

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Chromosome abnormalities in Burkitt lymphoma and Burkitt-type acute lymphoblastic leukemiakshop in 1981, the best known associations of this type were assembled for publication as a committee report [1]. A greatly enlarged compilation of similar data has appeared as a result of the Seventh Human Gene Mapping Workshop held in 1983 [2]. As many as 54 recurrent associations between a break

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