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Titlebook: Male Hypogonadism; Basic, Clinical, and Stephen J. Winters (Chief) Book 20041st edition Springer Science+Business Media New York 2004 Gonad

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發(fā)表于 2025-3-28 18:08:50 | 只看該作者
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發(fā)表于 2025-3-28 22:28:37 | 只看該作者
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發(fā)表于 2025-3-28 22:55:04 | 只看該作者
Normal and Delayed Puberty,thic), which is usually accompanied by a delay in height growth. Various endocrine disorders, causing hypogonadotropic or hypergonadotropic hypogonadism, must also be considered in boys with pubertal delay.
44#
發(fā)表于 2025-3-29 03:18:17 | 只看該作者
Cryptorchidism,ns about cryptorchidism in . in 1786, he not only noted that testes normally descend during the last 3 mo of gestation but also noted the conundrum that abnormal testes may fail to descend, whereas, conversely, failure to descend may result in abnormal testes.
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發(fā)表于 2025-3-29 10:39:08 | 只看該作者
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發(fā)表于 2025-3-29 12:53:12 | 只看該作者
Environmental Causes of Testicular Dysfunction,n was diagnosed as being altered by some environmental factor. Does this mean that such causes are rare or does it mean that they go unrecognized? Unfortunately, we probably still lack sufficient understanding to definitively answer this question, but hopefully this chapter will address it.
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發(fā)表于 2025-3-29 16:07:45 | 只看該作者
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發(fā)表于 2025-3-29 21:54:06 | 只看該作者
Book 20041st editionunction. Like other volumes in the Contemporary Endocrinology series, the goal of Male Hy- gonadism: Basic, Clinical, and Therapeutic Principles is to link current knowledge of basic biology to the practice of medicine. The development of new methods for testost- one replacement has substantially in
49#
發(fā)表于 2025-3-30 00:27:09 | 只看該作者
Neuroendocrine Control of Testicular Function,s maintained at a proper set point by the negative feedback effects of testicular steroids and inhibin-B. Testicular function is also influenced by multiple internal and external environmental factors.
50#
發(fā)表于 2025-3-30 05:07:08 | 只看該作者
Hypogonadism in Males With Congenital Adrenal Hyperplasia,e, or the protein StAR (intracellular cholesterol transport protein) lead to CAH. The most frequent cause of CAH (more than 90% of all cases) is 21-hydroxylase deficiency. This deficiency results in cortisol and aldosterone deficiency and an overproduction of 17-hydroxyprogesterone and androstenedione, leading to androgen excess.
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