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Titlebook: Male Hypogonadism; Basic, Clinical and Stephen J. Winters,Ilpo T. Huhtaniemi Book 2017Latest edition The Editor(s) (if applicable) and The

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發(fā)表于 2025-3-21 16:28:08 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Male Hypogonadism
副標(biāo)題Basic, Clinical and
編輯Stephen J. Winters,Ilpo T. Huhtaniemi
視頻videohttp://file.papertrans.cn/622/621916/621916.mp4
概述A revised and updated edition of the classic 2004 text on the etiology, diagnosis and treatment of male hypogonadism.Discusses both normal and abnormal testicular function and hypogonadism due to gene
叢書(shū)名稱(chēng)Contemporary Endocrinology
圖書(shū)封面Titlebook: Male Hypogonadism; Basic, Clinical and  Stephen J. Winters,Ilpo T. Huhtaniemi Book 2017Latest edition The Editor(s) (if applicable) and The
描述Now in a revised second edition, this comprehensive text covers all aspects of male hypogonadism from the basic science to clinical management, comprehensively explaining and applying new insights to the treatment of hypogonadal men. Chapters covering neuroendocrine control of testicular function, Leydig cell function, spermatogenesis, and normal and delayed puberty open the book. The focus then turns to the pathophysiology and treatment of hypogonadism and other forms of testicular dysfunction, such as Klinefelter syndrome, cryptorchidism, and disorders of the pituitary, as well as reproductive and endocrine consequences of cancer treatment, environmental factors, obesity and aging. Next are chapters that describe the available options for androgen replacement therapy, and the outcomes when men with hypogonadism of various causes are treated with testosterone, as well as a chapter devoted to current approaches to stimulating spermatogenesis in gonadotropin-deficient men.?.Highly practical and updated with the latest available data, this second edition of?.Male Hypogonadism: Basic, Clinical and Therapeutic Principles?.cogently presents a large body of scientific information on male
出版日期Book 2017Latest edition
關(guān)鍵詞Aging-related hypoandrogenemia; Androgen replacement therapy; Congenital hypogonadotropic hypogonadism
版次2
doihttps://doi.org/10.1007/978-3-319-53298-1
isbn_softcover978-3-319-85119-8
isbn_ebook978-3-319-53298-1Series ISSN 2523-3785 Series E-ISSN 2523-3793
issn_series 2523-3785
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

書(shū)目名稱(chēng)Male Hypogonadism影響因子(影響力)




書(shū)目名稱(chēng)Male Hypogonadism影響因子(影響力)學(xué)科排名




書(shū)目名稱(chēng)Male Hypogonadism網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱(chēng)Male Hypogonadism網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱(chēng)Male Hypogonadism被引頻次




書(shū)目名稱(chēng)Male Hypogonadism被引頻次學(xué)科排名




書(shū)目名稱(chēng)Male Hypogonadism年度引用




書(shū)目名稱(chēng)Male Hypogonadism年度引用學(xué)科排名




書(shū)目名稱(chēng)Male Hypogonadism讀者反饋




書(shū)目名稱(chēng)Male Hypogonadism讀者反饋學(xué)科排名




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Normal and Delayed Puberty, by the activation of the hypothalamic–pituitary–gonadal axis after the break that restrains this axis during the majority of childhood is released. This review will cover the clinical assessment of puberty, variation in the timing of puberty, and the complex factors that regulate the onset and timi
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Male Hypogonadism Resulting from Mutations in the Genes for the Gonadotropin Subunits and Their Recm. No germ line mutations of CGA have been reported, apparently because of the incompatibility of pregnancy maintenance in the absence of hGG. Five inactivating LHB mutations have been described in men with normal prenatal masculinization but arrested pubertal development. The three men so far descr
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發(fā)表于 2025-3-22 19:49:52 | 只看該作者
Hypogonadism in Males with Congenital Adrenal Hyperplasia,quent cause of impaired fertility. Their location adjacent to the testicular mediastinum, and their steroid-producing properties, may interfere with spermatogenesis and Leydig cell function. It is sometimes accompanied by hypergonadotropic hypogonadism. Hypogonadotropic hypogonadotropism is a second
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Klinefelter Syndrome and Other Forms of Primary Testicular Failure, least one supernumerary X chromosome resulting in a 47, XXY genotype. KS can present in childhood, adolescence or adulthood with varying degrees of hypogonadism, gynecomastia, very small testes and azoospermia. In adults, FSH and LH levels are elevated, and testosterone levels are low or low normal
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