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Titlebook: Lung Cancer; Volume 1: Molecular Barbara Driscoll Book 2003 Springer Science+Business Media New York 2003

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發(fā)表于 2025-3-21 18:35:15 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Lung Cancer
副標(biāo)題Volume 1: Molecular
編輯Barbara Driscoll
視頻videohttp://file.papertrans.cn/590/589070/589070.mp4
概述Includes supplementary material:
叢書(shū)名稱(chēng)Methods in Molecular Medicine
圖書(shū)封面Titlebook: Lung Cancer; Volume 1: Molecular Barbara Driscoll Book 2003 Springer Science+Business Media New York 2003
描述The Methods in Molecular Medicine series is intended as a resource for both novice and experienced investigators attempting to diversify their tech- logical base in research. Lung Cancer: Volume 1: Molecular Pathology Me- ods and Reviews presents an overview of the current status of assays employed to detect and characterize the multitude of pathologies that contribute to the development of this deadly disease. As with all volumes in the Methods in Molecular Medicine series, the reader should find that each methods-based chapter provides clear instructions for the performance of various protocols, supplemented by additional technical notes that provide valuable insight. These notes are designed to enable the reader to acquire the techniques described with a proficiency not easily achieved by re- ing standard method formats. No volume can exhaustively cover every aspect of biological research and there will be gaps in this endeavor that one or another research group will id- tify. Each section herein could readily be expanded into a book in its own right. However, I have sought to include a spectrum of techniques that should allow for the acquisition of key skills in each area cover
出版日期Book 2003
版次1
doihttps://doi.org/10.1385/1592593232
isbn_softcover978-1-4899-3912-8
isbn_ebook978-1-59259-323-1Series ISSN 1543-1894 Series E-ISSN 1940-6037
issn_series 1543-1894
copyrightSpringer Science+Business Media New York 2003
The information of publication is updating

書(shū)目名稱(chēng)Lung Cancer影響因子(影響力)




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Abrogation of the RB-p16Tumor Suppressor Pathway in Human Lung Cancern the . gene, with concomitant loss of the second allele-the prototypic mechanism of tumor suppressor gene inactivation. This occurs in almost all small cell lung cancers (SCLC), as well as in 15-25% of non-small cell lung cancers (NSCLC) (.-..
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Sensitive Detection of Hypermethylated p16INK4a Alleles in Exfoliative Tissue Materialation of otherwise nonmethylated genes is the active component of functional disturbance in cancer (.-.. Methylated islands will recruit special methyl-binding proteins and in conjunction with histone deacetylases are then thought to form repressive chromatin states around the promoter regions, lead
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Detection of the Transcripts and Proteins for the Transforming Growth Factor-β Isoforms and Receptorand functional analyses have demonstrated that TGF-β RI and TGF-β RII are serine/threonine kinases that are essential elements for TGF-β-mediated signal transduction (.,.. The cooperativity of TGF-β RI and TGF-β RII in signal transduction suggests that loss of functional TGF-β RI and/or TGF-β RII ex
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Ignacio I. Wistuba,Adi F. Gazdarin Rio de Janeiro, Brazil, was chosen be- cause it was in the homeland of Sergio Porto and provided an opportunity for his friends, colleagues, and countryrnen to pay hornage to hirn. The setting on Copacabana Beach afforded access to the lovely beaches, restaurants, and nightlife of one of the most
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