Titlebook: Liver Diseases; A Multidisciplinary Florentina Radu-Ionita,Nikolaos T. Pyrsopoulos,Eca Textbook 2020 Springer Nature Switzerland AG 2020 l

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Aviary

Liver Histologyely moving from vessel to perisinusoidal space of Disse facing the hepatocytes. In the Disse’s space, hepatic stellate cells, storing vitamin A, localize in a quiescent phenotype and they activate in response to chronic hepatic damage. The sinusoid endothelial cells and the hepatic stellate cells ar

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Molecular Basis of Fibrogenesis and Angiogenesis During Chronic Liver Disease: Impact of TGF-β and Vate latent form TGF-β, resulting in the enhancement of LC, suggesting a crosstalk between pathogenic angiogenesis and fibrosis. In this chapter, we would like to focus on the potential linkage of VEGF-based angiogenesis with TGF-β-enhanced fibrogenesis for understanding of LC-associated pathogenic p

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Intact

outskirts

CHIDE

Liver Ischaemia-Reperfusion Injuryllenge of IR injury is greater in liver transplantation, particularly with the increase in the use of marginal donors which are associated with increased risk of early allograft dysfunction and graft failure as well as biliary complications. Injury prevention starts at the donation process and conti

deriver

Parasitic Liver Diseasesressive and even lethal in some cases. Thus, diagnosis of the liver parasitosis mandates a high index of clinical suspicion before laboratory testing. In this chapter, the liver involvement in some of the clinically-relevant parasitic infections will be reviewed. A basic knowledge about the key clin

Sinus-Rhythm

Viral Hepatitis Btimately clear HBsAg at age 80. Those patients unable to clear HBV replication smoothly have increased risk of chronic hepatitis, liver cirrhosis, and hepatocarcinogenesis. Current therapies decrease hepatic decompensation and increase survival rate. However, the sustained virologic response rate is