Titlebook: Lipoprotein Metabolism and Atherogenesis; Toru Kita (Professor),Masayuki Yokode (Assistant P Conference proceedings 2000 Springer-Verlag T

頌歌

書目名稱Lipoprotein Metabolism and Atherogenesis影響因子(影響力)




書目名稱Lipoprotein Metabolism and Atherogenesis影響因子(影響力)學科排名




書目名稱Lipoprotein Metabolism and Atherogenesis網(wǎng)絡公開度




書目名稱Lipoprotein Metabolism and Atherogenesis網(wǎng)絡公開度學科排名




書目名稱Lipoprotein Metabolism and Atherogenesis被引頻次




書目名稱Lipoprotein Metabolism and Atherogenesis被引頻次學科排名




書目名稱Lipoprotein Metabolism and Atherogenesis年度引用




書目名稱Lipoprotein Metabolism and Atherogenesis年度引用學科排名




書目名稱Lipoprotein Metabolism and Atherogenesis讀者反饋




書目名稱Lipoprotein Metabolism and Atherogenesis讀者反饋學科排名

冒煙

Role Of Lipid Concentrations and Apolipoproteins In Altering ApoB Metabolism and Promoting Coronary inked to coronary events are HDL cholesterol and triglycerides. Baseline triglycerides predict coronary risk in the major statin trials. In CARE and 4S, the triglyceride concentrations during treatment with pravastatin or simvastatin were associated with coronary events. Additional lipid mechanisms

nurture

Long-term Effect of LDL-Apheresis on Familial Hypercholesterolemia Homozygotest in South Africa showed atorvastatin being capable of suppressing cholesterol synthesis and useful in reducing the rebound in patients with homozygous FH under apheresis treatment. Another clinical trial carried out in Japan also showed the efficacy of the statin in such patients, suggesting that t

信條

Diabetes and Atherosclerosis (5.2±0.1 and 3.2±0.6 ng/mg cell protein, respectively; p<0.02). The amount of oxidized LDL associated to cells grown in 400 mg/dl of glucose was markedly increased compared with the cells grown in 200 mg/dl of glucose. Our results suggesetd that an suitable glucose concentration (400 mg/dl) associa

波動

Molecular Analysis of Apolipoprotein A-I and E Mutants in Japann of apoE gene is etiologically related to LPG. However, our cases indicate that other genetic or environmental factors seem to be necessary for the development of LPG. Identification of naturally occurring apolipoprotein variants allows understanding the role of apolipoproteins and the mechanism of

罵人有污點

Novel mutations of the LDL Receptor Gene in Familial Hypercholesterolemia Pedigrees in Hokkaido stretch at 1774-8 (codons 571-572), causing a frameshift, in six members of family D; (5) a one-base deletion of T at nucleotide 1963-4 (codon 634), causing a frameshift, in three members of family E. Through the molecular genetic approach a total of 28 individuals were diagnosed unequivocally as h

預定

Preheparin Lipoprotein Lipase Mass Might Reflect the Amount of Working Lipoprotein Lipase in the Whos augmented, and was also enhanced in the situations in which insulin is functioning well. Then, it might be concluded that preheparin LPL mass can be a noble indicator for the amount of LPL functioning in a whole body, even though indirectly.

情節(jié)劇

培養(yǎng)

Evidence of linkage in familial combined hyperlipidemia to chromosome 1q21-q23y avoid problems associated with each of the analysis methods alone. No evidence for linkage existed between FCHL and intragenic markers of the candidate genes studied. However, one flanking marker for the apolipoprotein A-II (apoA2) gene resulted in a statistically significant lod score of 3.50 on

Asymptomatic

Effect of troglitazone on plasma lipid metabolism and lipoprotein lipaseplasma glucose and glycohemoglobin Alc were 216 ± 34, 160±84, 57± 19, 145±30 mg/dl and 7.8 ± 1.6 %. Four weeks after treatment, those levels were 209 ±36, 105 ±27 ( p=0.004 ), 63 ± 19 ( p=0.02 ) mmol/l, 139±41 mg/dl and 7.3 ±0.6 % ( p=0.01), respectively. The postheparin plasma LPL mass increased fr