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Titlebook: Lipoprotein Deficiency Syndromes; Aubie Angel,Jiri Frohlich Book 1986 The Editor(s) (if applicable) and The Author(s), under exclusive lic

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書目名稱Lipoprotein Deficiency Syndromes
編輯Aubie Angel,Jiri Frohlich
視頻videohttp://file.papertrans.cn/587/586903/586903.mp4
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Lipoprotein Deficiency Syndromes;  Aubie Angel,Jiri Frohlich Book 1986 The Editor(s) (if applicable) and The Author(s), under exclusive lic
描述Current interest in lipoprotein deficiency states stems from the growing realization of their importance in the etiology of premature coronary heart disease. While hypercholesterolemia and coronary heart disease risk are strongly correlated in their etiologic relationship, it is becoming equally clear that deficiencies in HDL, whether congenital or acquired, also enhance the risk for the future development of coronary atherosclerosis. This has led to renewed attention to the lipid hypothesis and realization of the fact that each lipoprotein class and apoprotein species has specific functions in the transport and cellular uptake of various lipids. It is a truism that a biochemical correlate of disease once identified is subsequently recognized with increasing frequency in clinical medicine. The story of HDL was no exception. Indeed hypoalphalipoproteinemia appears to be a disease of high prevalence approaching and perhaps even exceeding that of familial hypercholesterolemia. Its clinical signifi- cance escaped our notice for many years largely due to a heavy emphasis on hypercholesterolemia and to difficulties in measuring HDL reliably.
出版日期Book 1986
關(guān)鍵詞Corona; Lipid; biology; development; enzyme; enzymes; etiology; lipoprotein; medicine; metabolism; molecular b
版次1
doihttps://doi.org/10.1007/978-1-4684-1262-8
isbn_softcover978-1-4684-1264-2
isbn_ebook978-1-4684-1262-8Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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HDL Binding to Human Adipocyte Plasma Membranes: Regional Variation in Omental and Subcutaneous Deporol in normal man (1,2). This adipose cholesterol pool is much expanded in obesity and in the massively obese state, well over half of total body cholesterol can be found in the fat tissue (1,2). Obesity is also commonly associated with a number of lipoprotein abnormalities. Increased synthesis and/
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發(fā)表于 2025-3-22 20:08:59 | 只看該作者
Clinical, Nutritional and Biochemical Consequences of Apolipoprotein B Deficiencyetabolic abnormality. We have continued to follow some 25 patients with the homozygous form of either ABL or hypobetalipoproteinemia. Our efforts are directed toward the clinical and biochemical evaluation of patients with particular stress on delineating changes in the neurological system, along wi
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Severe Hypoalphalipoproteinemia Induced by a Combination of Probucol and Clofibrateofibrate (2 g/day) prompted a review of all cases treated by this combination at our lipid clinic. Hypoalphalipoproteinemia (HDL-C >15 mg/dL) developed in 19 of 28 (70%) hyperlipidemic subjects who received this combination for an average of 1.5 years. This effect was sustained and reversible; it di
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發(fā)表于 2025-3-23 06:19:21 | 只看該作者
Control of Plasma HDL Levels After Plasmapheresisnce then it has become increasingly clear that twice monthly exchanges with plasma protein fraction (PPF) provide a safe and effective means of treating this potentially fatal condition. Several patients have undergone this regimen for over 9 years without any significant acute complications or long
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