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Titlebook: Lipid Transfer in Lipoprotein Metabolism and Cardiovascular Disease; Xian-Cheng Jiang Book 2020 The Editor(s) (if applicable) and The Auth

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發(fā)表于 2025-3-30 09:58:22 | 只看該作者
Liqing Yu,Yi Li,Alison Grisé,Huan Wangtheoretical results or develop algorithms. In the second half of the past century various generalizations of convex functions have been introduced. We mention here the early work by de Finetti [54], Fenchel [65], Arrow and Enthoven [5], Mangasarian [142], Ponstein [203] and Karamardian [109]. Usuall
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發(fā)表于 2025-3-31 00:41:44 | 只看該作者
Circadian Clock Regulation on Lipid Metabolism and Metabolic Diseases,via regulation of circadian clock genes or clock-controlled genes in peripheral tissue), which lead to a variation in plasma phospholipids and tissue phospholipids. Circadian clock genes affect the regulation of transporters and proteins included in the regulation of phospholipid metabolism. These g
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發(fā)表于 2025-3-31 03:57:24 | 只看該作者
ABC Transporters, Cholesterol Efflux, and Implications for Cardiovascular Diseases,ses in these cells, particularly in lesional macrophages. ABCA1 and ABCG1 are highly expressed in macrophages, particularly in response to cholesterol accumulation, and are essential in maintenance of cholesterol homeostasis. Functional deficiency of ABCA1 and ABCG1 in macrophage markedly increases
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發(fā)表于 2025-3-31 05:28:40 | 只看該作者
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發(fā)表于 2025-3-31 20:32:53 | 只看該作者
Rare Diseases Related with Lipoprotein Metabolism,widely used for lipid management and decreasing cardiovascular disease risks. Translating the clinical extreme phenotypes into laboratory bench work has turned out to be the first important step in the paradigm conducting translational and precise medical research. Here we review the main categories
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發(fā)表于 2025-4-1 00:23:31 | 只看該作者
CGI-58: Versatile Regulator of Intracellular Lipid Droplet Homeostasis,d glucose metabolism, energy balance, insulin signaling, inflammatory responses, and thermogenesis. Recent studies have shown that CGI-58 regulates the pathogenesis of common metabolic diseases in a tissue-specific manner. Future studies are needed to molecularly define ATGL-independent functions of
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