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Titlebook: Lipid Metabolism in the Healthy and Disease Heart; Ger J. Vusse,Hans Stam Book 1992 Springer Science+Business Media Dordrecht 1992 ATP.Lip

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發(fā)表于 2025-3-25 03:21:44 | 只看該作者
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發(fā)表于 2025-3-25 08:41:23 | 只看該作者
Long term Incubation of Cardiac Myocytes with Oleic Acid and Very-low Density Lipoprotein Reduces Hould be released into the medium when cardiac myocytes were incubated with heparin. Incubation of cardiac myocytes with VLDL, or the products of TG breakdown, oleic acid or 2-monoolein, did not increase LPL activity in the medium. However, incubation of cardiac myocytes with either VLDL or oleic aci
23#
發(fā)表于 2025-3-25 15:28:36 | 只看該作者
Carnitine Palmitoyltransferase in the Heart is Controlled by a Different Mechanism than the Hepaticaspect of the regulatory mechanism for . fatty acid oxidation. Inhibition of myocardial carnitine palmitoyltransferase by malonyl-CoA may play an important role in regulation of fatty acid oxidation in the heart, but there has been a discrepancy in data relating to changes in malonyl-CoA sensitivity
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發(fā)表于 2025-3-26 03:15:04 | 只看該作者
Occurrence and Functions of the Phosphatidylinositol Cycle in the Myocardium,ules and which involves the hydrolysis of a quantitatively minor phospholipid, phosphatidylinositol 4,5-bisphosphate (PtdIns(4,5)P.) by a Ptdlns-specific phospholipase C (PLC). The evidence for the existence of receptor-mediated GTP binding protein-coupled PLC in myocardium and its possible function
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