Titlebook: Lipid Metabolism in Normoxic and Ischemic Heart; Ger J. Vusse Book 1989 Kluwer Academic Publishers 1989 Lipoprotein.assessment.attention.d

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Kees Schoonderwoerd,Ton van der Kraaij,Willem C. Hülsmann,Hans Stamall the incentives in our eleven countries. That would go beyond the scope of this monograph. Instead we have chosen an overall survey of problems and solutions illustrated by examples from the various countries. We limit ourselves to tax incentives I–V in the catalogue of Chapter IV, thus omitting

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Myocardial fatty acid homeostasis,on unraveling the complexity of fatty acid homeostasis in the normal and diseased heart. The broad interests in cardiac lipid metabolism had led to the organization of the 1. International Symposium on lipid Metabolism in the Normoxic and Ischemi Heart in Rotterdam, The Netherlands, in 1986. The pro

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Effect of taxol on the heparin-induced secretion of lipoprotein lipase from cardiac myocytes,slower rate of release (10–60 min). Reducing the incubation temperature from 37° C to 23° C inhibited the slow phase of secretion, but had no effect on the rapid phase. Similarly, taxol, a microtubule-stabilizing drug, selectively reduced the slow phase of LPL release, without influencing the rapid

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Transmembrane transport of fatty acids in the heart,presented to the plasma membrane of cardiomyocytes as albumin complexes within the plasma. Since albumin is not taken up by the cells, it was postulated that specific high affinity binding sites at the sarcolemma may mediate the dissociation of fatty acids from the albumin molecules, before they are