Titlebook: Lipid Metabolism in Normoxic and Ischemic Heart; Ger J. Vusse Book 1989 Kluwer Academic Publishers 1989 Lipoprotein.assessment.attention.d

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Intracellular transport of lipids, of H-FABP in several other tissues, including red skeletal muscle, mammary gland and kidney. Beside long-chain fatty acids FABP binds with similar affinity also fatty acyl-CoA and acyl-L-carnitines. In heart the latter compound may be the primary ligand, since normoxic acyl-L-carnitine levels are s

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Modeling of palmitate transport in the heart,passive exchange. Inside the myocyte palmitate is again bound to the fatty acid binding protein which buffers the free palmitate concentration, facilitates diffusion, and may facilitate further intracellular reactions.

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Degradation of phospholipids and triacylglycerol, and accumulation of fatty acids in anoxic myocarde A. rather than A. is active in autolyzing cardiac tissue. The accumulation of fatty acids corresponded with the loss of triacylglycerol and phospholipids from the tissue during 120 minutes of autolysis.

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Subcellular distribution, molecular dynamics and catabolism of plasmalogens in myocardium,ties of probes attached to their .-2 aliphatic constituents. Furthermore, since free radical oxidation of myocardial lipid constituents occurs during myocardial ischemia and reperfusion, we demonstrated that .O. mediated oxidation of plasmenylcholine resulted in the generation of several products wh

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Phospholipase C-evoked glycerol release in energy depleted rat myocardial cells,genase in energy depleted myocytes. Normal cells, on the other hand, were apparently not affected by PLC. These data suggest that PLC selectively attacks energy depleted and/or structurally damaged myocytes. This could well enhance the breakdown of the natural barrier between the extra- and intracel

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