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Titlebook: Klinische Strahlenbiologie; Thomas Herrmann Book 1978 VEB Gustav Fischer Verlag Jena 1978 Radiologie.Strahlenbiologie.Strahlenschutz.Strah

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21#
發(fā)表于 2025-3-25 06:32:50 | 只看該作者
22#
發(fā)表于 2025-3-25 08:38:05 | 只看該作者
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發(fā)表于 2025-3-25 12:13:28 | 只看該作者
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發(fā)表于 2025-3-25 16:45:04 | 只看該作者
https://doi.org/10.1007/978-3-642-95963-9Radiologie; Strahlenbiologie; Strahlenschutz; Strahlentherapie; Strahlun; Strahlung; Radiaton Oncology
25#
發(fā)表于 2025-3-25 21:12:21 | 只看該作者
, numerous tumor suppressor pathways are commonly inactivated including p53, Rb/CDKN2A, NOTCH, and TGFβ/SMAD, as are a host of epigenetic regulatory enzymes. Deregulation of these genetic pathways alters key cellular processes within the stratified squamous epithelium including proliferation, termin
26#
發(fā)表于 2025-3-26 04:01:40 | 只看該作者
Thomas Herrmannted to a few loci, most importantly .., . and ., which gave evidence of disease relevance by statistically significant associations and replication in large homogenous study populations. These genes are largely located in regulatory networks that integrate interactions of the metabolic and immune sy
27#
發(fā)表于 2025-3-26 07:26:03 | 只看該作者
28#
發(fā)表于 2025-3-26 11:28:37 | 只看該作者
Thomas Herrmannulence give a wide variety of phenotypes: defects in known virulence factors have been found, thereby validating the method; defects have also been found in orthologs with predicted functions, and in some genes whose functions cannot be predicted from databases. A general strategy and a simple scena
29#
發(fā)表于 2025-3-26 14:41:39 | 只看該作者
30#
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