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Titlebook: Klinische Endokrinologie; Ein Lehrb. f. Arzte Arthur Jores Book 19391st edition Springer-Verlag Berlin Heidelberg 1939 Endokrinologie.Klin

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11#
發(fā)表于 2025-3-23 13:09:27 | 只看該作者
Arthur Jores protein and the role of ligand interaction in T-cell activation remains obscure. We have shown that the consequences of CTLA-4 engagement is the delivery of a negative signal for T-cell response. Thus, the binary view of co-stimulation via B7 should be expanded to include a third signal generated b
12#
發(fā)表于 2025-3-23 14:36:09 | 只看該作者
13#
發(fā)表于 2025-3-23 18:31:01 | 只看該作者
Arthur Joresdiseases (3). All hematopoietic cell types are of major interest in this context, since the modification of the hematopoietic stem cell population may potentially give rise to a completely transgenic hematopoiesis with the potential to cure genetic disorders or fight severe chronic infections, and t
14#
發(fā)表于 2025-3-23 22:59:53 | 只看該作者
15#
發(fā)表于 2025-3-24 03:56:37 | 只看該作者
Arthur Jorese modification of the hematopoietic stem cell population may potentially give rise to a completely transgenic hematopoiesis with the potential to cure genetic disorders or fight severe chronic infections, and t978-1-61737-866-9978-1-59745-409-4Series ISSN 1064-3745 Series E-ISSN 1940-6029
16#
發(fā)表于 2025-3-24 08:33:23 | 只看該作者
17#
發(fā)表于 2025-3-24 12:03:55 | 只看該作者
e modification of the hematopoietic stem cell population may potentially give rise to a completely transgenic hematopoiesis with the potential to cure genetic disorders or fight severe chronic infections, and t978-1-61737-866-9978-1-59745-409-4Series ISSN 1064-3745 Series E-ISSN 1940-6029
18#
發(fā)表于 2025-3-24 16:43:32 | 只看該作者
cience. When functioning properly, the immune system prevents us from succumbing to the microbial world; however, when it goes awry, it can mediate a variety of insidious pathological disease. These include classical autoimmune diseases such as lupus erythematosus, arthritis, and allergy, or conditi
19#
發(fā)表于 2025-3-24 19:09:43 | 只看該作者
Arthur Jores (MHC) molecules with the . (TCR). A second antigen-independent signal is generated through interactions of CD28 with members of the B7 family: B7-1 (CD80) and B7-2 (CD86). Antibodies to CD28 or B7 transfectants are both able to supply this second signal in vitro. Co-stimulation has also been demons
20#
發(fā)表于 2025-3-24 23:17:16 | 只看該作者
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