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Titlebook: Kindling 4; Juhn A. Wada Book 1990 Plenum Press, New York 1990 Aspartat.adenosine.amino acid.anatomy.brain.brainstem.cortex.development.el

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發(fā)表于 2025-3-21 17:55:10 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Kindling 4
編輯Juhn A. Wada
視頻videohttp://file.papertrans.cn/544/543013/543013.mp4
叢書名稱Advances in Behavioral Biology
圖書封面Titlebook: Kindling 4;  Juhn A. Wada Book 1990 Plenum Press, New York 1990 Aspartat.adenosine.amino acid.anatomy.brain.brainstem.cortex.development.el
描述This is the proceedings of the fourth in a series of inter- national gatherings on kindling held on the University of British Columbia campus, June 15-17, 1989. Since the last gath- ering in 1985, kindling continues to attract an ever-increasing number of investigators as reflected by the phenomenal increase in the number of kindling-related research reports. No other acute or chronic model has been exploited so extensively using electrophysiological, pharmacological, biochemical and behav- ioural approaches involving a variety of brain sites and ani- mal species. The continuing search, during the past few years, for the mechanism underlying the enduring change induced by kindling is beginning to shed some light on aspects of its molecular basis and to suggest the future direction of research. The late Graham Goddard, the original kindler, would have been delighted with this development. We were all shocked and saddened by his tragic death, but his spirit is very much alive among all of us who gathered together to share new information and collectively reassess the present state of knowledge at this symposium. I must say that we miss him very, very much. We know he is happy in his
出版日期Book 1990
關(guān)鍵詞Aspartat; adenosine; amino acid; anatomy; brain; brainstem; cortex; development; electroencephalography (EEG
版次1
doihttps://doi.org/10.1007/978-1-4684-5796-4
isbn_softcover978-1-4684-5798-8
isbn_ebook978-1-4684-5796-4Series ISSN 0099-6246
issn_series 0099-6246
copyrightPlenum Press, New York 1990
The information of publication is updating

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Book 1990-17, 1989. Since the last gath- ering in 1985, kindling continues to attract an ever-increasing number of investigators as reflected by the phenomenal increase in the number of kindling-related research reports. No other acute or chronic model has been exploited so extensively using electrophysiolog
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Mechanisms of Kindling in Developing Animals,AD) in the absence of any behavioral manifestations. Gradually, as the behavioral manifestations become more apparent, the ADs intensify and increase in duration. Once established, the kindling effect persists for several months suggesting that it produces permanent changes in the brain (2).
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Kindling as a Model of Long-Lasting Plasticity: New Perspectives in the Search for Molecular Mechano the analysis of antiepileptic drugs and the study of the neurobiological basis of epileptogenesis. Now, the overall tendency is to reconcile both aspects in again considering kindling as a model of plasticity. A paper on this subject is presented in the book Kindling 3 (27).
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Metabolic Maps of Kindling: A Sequential Double-Label Deoxyglucose Study,h disseminated functional alterations must relate not only to the seizure phenomenon but also to the secondary brain dysfunction manifested in some epileptics. However, we do not yet have a comprehensive knowledge of the anatomical system of kindling and induced alterations.
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