Titlebook: Kaposi Sarcoma Herpesvirus: New Perspectives; Chris Boshoff,Robin A. Weiss Book 2007 The Editor(s) (if applicable) and The Author(s), unde

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The Rta/Orf50 Transactivator Proteins of the Gamma-Herpesviridae,sviruses including Epstein-Barr virus (EBV), rhesus rhadinovirus (RRV), herpesvirus saimiri(HVS), and murineherpesvirus 68 (MHV-68). Each Rta/Orf50 homologue of each gammaherpesvirus plays a pivotal role in the initiation of viral lytic gene expression and lytic reactivation from latency. Here we di

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Structure and Function of Latency-Associated Nuclear Antigen,ed malignancies. LANA is a large (222–234 kDa) nuclear protein that interacts with various cellular as well as viral proteins. LANA has been classified as an oncogenic protein as it dysregulates various cellular pathways including tumor suppressor pathways associated with pRb and p53 and can transfo

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The KSHV and Other Human Herpesviral G Protein-Coupled Receptors,al cell malignancy responsible for significant morbidity and mortality worldwide. Over time, KSHV has pirated many human genes whose products regulate angiogenesis, inflammation, and the cell cycle. One of these encodes for a mutated G protein-coupled receptor (GPCR) that is a homologue of the human

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Regulation of KSHV Lytic Gene Expression,roduct, replication and transcription activator (RTA), has been identified as the molecular switch for initiation of the lytic gene expression program from latency. Here we review progress on two key questions: how RTA gene expression is controlled by viral proteins and cellular signals and how RTA

ensemble

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KSHV After an Organ Transplant: Should We Screen?,ter than in the general population, and its occurrence is associated with immunosuppressive therapy. The reported incidence of posttransplant KS ranges from 0.5% to 5%, depending on the patient’s country of origin and the type of organ received, mainly after renal transplantation. Posttransplant KS

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