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Titlebook: Invertebrate Cytokines and the Phylogeny of Immunity; Facts and Paradoxes Alain Beschin,Werner E. G. Müller Book 2004 Springer-Verlag Berli

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書(shū)目名稱Invertebrate Cytokines and the Phylogeny of Immunity
副標(biāo)題Facts and Paradoxes
編輯Alain Beschin,Werner E. G. Müller
視頻videohttp://file.papertrans.cn/475/474721/474721.mp4
概述Most recent summary of the uses of molecular biology as a tool for studies of cytokines in invertebrates.Cytokines are molecules involved in the signaling pathway of wounding and immune responses
叢書(shū)名稱Progress in Molecular and Subcellular Biology
圖書(shū)封面Titlebook: Invertebrate Cytokines and the Phylogeny of Immunity; Facts and Paradoxes Alain Beschin,Werner E. G. Müller Book 2004 Springer-Verlag Berli
描述.Based on the assumption that invertebrates as well as?vertebrates possess factors regulating hematopoiesis, response to infection or wounding, studies dealing with the evolution of immunity have focused on the isolation and characterization of putative cytokine-related molecules from invertebrates. Until recently, most of our knowledge of cytokine- and cytokine receptor-like molecules in invertebrates has relied on functional assays and similarities at the physicochemical level. As such, a phylogenetic relationship between invertebrate cytokine-like molecules and invertebrate counterparts could not be convincingly demonstrated. .In the present book, recent studies demonstrating cytokine-like activities and related signaling pathways in invertebrates are critically reviewed, focusing on findings from molecular biology and taking advantage of the completion of the genome from the fly Drosophila and the worm Caenorhabditis elegans..
出版日期Book 2004
關(guān)鍵詞Drosophila; biology; cytokine; cytokines; evolution; genome; hematopoiesis; immunity; infection; invertebrate
版次1
doihttps://doi.org/10.1007/978-3-642-18670-7
isbn_softcover978-3-642-62236-6
isbn_ebook978-3-642-18670-7Series ISSN 0079-6484 Series E-ISSN 2197-8484
issn_series 0079-6484
copyrightSpringer-Verlag Berlin Heidelberg 2004
The information of publication is updating

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