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Titlebook: Intracellular Regulation of Ion Channels; Martin Morad,Zalman Agus Conference proceedings 1992 Springer-Verlag Berlin Heidelberg 1992 ATP.

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發(fā)表于 2025-3-23 12:56:45 | 只看該作者
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發(fā)表于 2025-3-23 16:32:18 | 只看該作者
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發(fā)表于 2025-3-23 18:31:51 | 只看該作者
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發(fā)表于 2025-3-23 22:11:25 | 只看該作者
Vaccinia Virus: A Novel Expression System for Receptors and Ion Channelsclones in foreign cells. Ideally they are re-introduced into cells whose intracellular milieu is similar to the protein’s natural environment; this increases the likelihood of correct biosynthesis, posttranslational modification, sorting, assembly, and targeting of the expressed proteins.
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發(fā)表于 2025-3-24 05:03:05 | 只看該作者
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發(fā)表于 2025-3-24 09:35:48 | 只看該作者
Receptor-Dependent Modulation of G Protein-Gated Muscarinic K+ Channel by Arachidonic Acid Metabolittimulatory manner. The pathway could be activated by various receptor agonists, such as phenylephrine (α.-adrenergic agonist) and platelet-activating factor. The arachidonic acid metabolite-mediated activation of the K. channel was independent from muscarinic ACh and adenosine receptors, which are d
17#
發(fā)表于 2025-3-24 11:44:13 | 只看該作者
18#
發(fā)表于 2025-3-24 17:55:10 | 只看該作者
Regulation of the ATP-Dependent K+ Channel of Muscle by ATP and pHtal and smooth muscle (Spruce, Standen & Stanfield, 1985; Burton, D?rstelmann & Hutter, 1988; Standen et al., 1989), as well as in other tissues. This paper will focus mainly on their properties and regulation in skeletal muscle.
19#
發(fā)表于 2025-3-24 21:51:56 | 只看該作者
Regulation of Cardiac ATP-Sensitive K+ Channelsvalues as high as 18 mM within 10 minutes (Kleber, 1984). [K.]. accumulation of this magnitude has profound effects on the electrophysiological properties of the ischemic region, and is a key factor leading to the development of lethal ventricular arrhythmias (Janse & Wit, 1989), the major cause of
20#
發(fā)表于 2025-3-25 01:46:32 | 只看該作者
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