Titlebook: Interstitial Fibrosis in Heart Failure; Francisco J. Villarreal Book 2005 Springer-Verlag New York 2005 gene therapy.heart.heart failure.m

忠誠

書目名稱Interstitial Fibrosis in Heart Failure影響因子(影響力)




書目名稱Interstitial Fibrosis in Heart Failure影響因子(影響力)學(xué)科排名




書目名稱Interstitial Fibrosis in Heart Failure網(wǎng)絡(luò)公開度




書目名稱Interstitial Fibrosis in Heart Failure網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Interstitial Fibrosis in Heart Failure被引頻次




書目名稱Interstitial Fibrosis in Heart Failure被引頻次學(xué)科排名




書目名稱Interstitial Fibrosis in Heart Failure年度引用




書目名稱Interstitial Fibrosis in Heart Failure年度引用學(xué)科排名




書目名稱Interstitial Fibrosis in Heart Failure讀者反饋




書目名稱Interstitial Fibrosis in Heart Failure讀者反饋學(xué)科排名

CHIDE

Pharmacology of G-Protein-linked Signaling in Cardiac Fibroblastscardiovascular diseases differ from “normal” CFs. Actions of hormones are likely much more complex . than ., as the example of . agonists makes clear. In addition, cross-talk amongst signaling pathways when cells are stimulated by multiple agonists complicates the picture and the overall responses.

BINGE

Ultrasonic Characterization of the Myocardiumce of myocardial fibrosis and how these measurements lay the foundation for the continuing development of clinically applicable noninvasive ultrasonic assessment techniques. Several studies were cited which show how ultrasonic propagation and scattering are directly related to fundamental physical p

PATRI

aphasia

certitude

Cardiac Mast Cells as Mediators of Ventricular Remodelingdiac chamber distension there is a release of atrial natriuretic peptide, which is known to cause mast cell degranulation (Figure 2). Secreted . tryptase and chymase are all capable of activating matrix metalloproteinases, which in turn are responsible for fibrillar collagen degradation. Since one o

紀(jì)念

ARENA

Pde5-Inhibitors

Biological and Functional Effects of Chronic Mechanical Support Induced by Left Ventricular Assist Dtechnology improves and we are able to avoid long-term mechanical failures and reduce the complications associated with the implantation of these devices. However, what has become clear is the failing heart, which appears to be “endstage,” may have the remarkable ability to recover at least at the c

SPECT