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Titlebook: Information Technologies and Mathematical Modelling. Queueing Theory and Applications; 16th International C Alexander Dudin,Anatoly Nazarov

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發(fā)表于 2025-3-30 10:51:35 | 只看該作者
Anatoly Nazarov,Tuan Phung-Duc,Svetlana Paulition. We keep to this original concept, as did the mathematicians when they called their nonmolecular computer parasites “viruses”, meaning nothing more than invisible replicating parasites that required the energy and informational systems of the host for their replication.
52#
發(fā)表于 2025-3-30 14:59:33 | 只看該作者
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發(fā)表于 2025-3-30 19:40:48 | 只看該作者
Ekaterina Lisovskaya,Svetlana Moiseeva,Michele Paganoilure in a fraction of, locally controlled patients, recent retrospective studies have demonstrated that local recurrence at the primary tumor site is associated with a significant increase in metastatic disease in nearly every type of tumor tested (F. et al. 1991a; L. et al. 1991b). Some investigat
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發(fā)表于 2025-3-30 22:55:04 | 只看該作者
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發(fā)表于 2025-3-31 01:03:17 | 只看該作者
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發(fā)表于 2025-3-31 07:52:03 | 只看該作者
57#
發(fā)表于 2025-3-31 11:05:54 | 只看該作者
Evsey Morozov,Lyubov Potakhina.) alone or fused to multiple copies of the murine C3d or a twenty-eight amino-acid peptide (P28) containing a minimum CR2 binding domain. Each immunogen was expressed from DNA plasmid in vivo or injected as purified recombinant protein. The fusion of the P28 peptide to Env. enhanced both humoral an
58#
發(fā)表于 2025-3-31 16:28:53 | 只看該作者
Ekaterina Pankratova,Mais Farkhadov,Erol Gelenbectivation, as evidenced by the binding and activation of C1 and generation of C4d. However, neither C1q-deficient serum nor a truncated form of gC1qR (gC1qRΔ74–96), supported complement activation. Taken together, the data strongly suggest that at sites of inflammation, such as vasculitis and athero
59#
發(fā)表于 2025-3-31 19:29:48 | 只看該作者
60#
發(fā)表于 2025-3-31 23:02:43 | 只看該作者
Maria Shklennik,Svetlana Moiseeva,Alexander Moiseevctivation, as evidenced by the binding and activation of C1 and generation of C4d. However, neither C1q-deficient serum nor a truncated form of gC1qR (gC1qRΔ74–96), supported complement activation. Taken together, the data strongly suggest that at sites of inflammation, such as vasculitis and athero
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