Titlebook: Inflammation and Cardiac Diseases; Giora Z. Feuerstein,Peter Libby,Douglas L. Mann Book 2003 Springer Basel AG 2003 Cardiology.atrial fibr

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書目名稱Inflammation and Cardiac Diseases影響因子(影響力)




書目名稱Inflammation and Cardiac Diseases影響因子(影響力)學(xué)科排名




書目名稱Inflammation and Cardiac Diseases網(wǎng)絡(luò)公開度




書目名稱Inflammation and Cardiac Diseases網(wǎng)絡(luò)公開度學(xué)科排名




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書目名稱Inflammation and Cardiac Diseases被引頻次學(xué)科排名




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書目名稱Inflammation and Cardiac Diseases年度引用學(xué)科排名




書目名稱Inflammation and Cardiac Diseases讀者反饋




書目名稱Inflammation and Cardiac Diseases讀者反饋學(xué)科排名

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Toll-like receptors and the cardiovascular systempatterns” (“PAMPs”), as for example lipopolysaccharides of bacteria or double-stranded RNA of viruses [.]. This review will focus on the toll-like receptor (TLR) family, a class of pattern recognition receptors of major importance [.].

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1422-7746 great value to researchers as well as cliniciansHeart failure research is a most active area of research in academic, industrial and government-sponsored research and receives intense clinical attention. The recent recognition that inflammation is a risk factor and prognostic factor for heart diseas

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Inflammation — A new frontier in cardiac disease and therapeuticstally an interlinked array of host defense mechanism that features:.“… localized protective response elicited by injury or destruction of tissue, which serves to destroy, dilute, to wall off (sequester) both the injurious agent and the injured tissue.” [., .].

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A biomarker of inflammation in cardiovascular diseaselevels [.]. However, levels of CRP increase markedly in response to trauma or disease and closelytrack the inflammatory burden of disease. Synthesis and release of CRP appears tobe largely controlled by the cytokine IL-6. As the role of inflammation in cardiacdisease gains greater acceptance, the si

Stress-Fracture

Inflammation and coronary artery diseases is now considered a complex inflammatory process in response to the retention of specific atherogenic lipoproteins in the arterial wall. In addition to the localized immune response, there is emerging evidence that systemic inflammatory markers may have a diagnostic value in predicting acute cardi

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The role of matrix metalloproteinases in LV remodeling following myocardial infarctionnction, it is becoming widely accepted that the ECM is a dynamic entity that interacts with cells and regulates cell phenotype. In this regard, the matrix profoundly affects cell behavior through signaling by integrins, which form direct contacts between cells and the ECM [.]. Second, proteoglycans