Titlebook: Inducible Enzymes in the Inflammatory Response; Derek A. Willoughby,Annette Tomlinson Book 1999 Springer Basel AG 1999 clinic.enzyme.infla

凹處

iNOS and COX-2 in atherosclerosis,n the Western world today [1, 2]. The disease is regulated by a large number of different factors which partly explains why the aetiology and pathogenesis of atherosclerosis remain incompletely defined. Since the discovery of prostacyclin in the 1970s [3] and nitric oxide in the 1980s [4] considerab

Infinitesimal

The role of the inducible enzymes cyclooxygenase-2, nitric oxide synthase and heme oxygenase in ang quiescent and endothelial cell turnover may be measured in terms of years. However, it is an essential component of a variety of normal functions such as embryogenesis, normal tissue growth and the menstrual cycle. It also plays a role in the pathology of a variety of disease states and this led Ju

Atmosphere

可觸知

Neuroinflammation,atomically tight interface between blood vessel wall and the neural parenchyma, the so called blood-brain (BBB) and blood-nerve barrier (BNB), respectively, form a strict separation between the systemic immune compartment and the nervous system, and that (b) other immunologic mechanisms such as the

Dictation

Inducible enzymes in inflammation: advances, interactions and conflicts,tion has focused on inducible nitric oxide synthase (iNOS), the enzyme which catalyses L-argininine to L-citrulline and nitric oxide (NO), and on the recently discovered second isoform of cyclooxygenase (COX-2), which converts arachidonic acid to bioac-tive prostanoids.

哺乳動物

文藝

The role of the inducible enzymes cyclooxygenase-2, nitric oxide synthase and heme oxygenase in angature; these include many of the chronic inflammatory diseases such as rheumatoid arthritis [2]. In these latter diseases, the neovasculature not only acts as a route for the increased nutrient supply required by the developing tissue, but also provides a greatly exaggerated area of activated endoth

游行

Neuroinflammation,egulating the immunologic machinery in the nervous system. The model in which an aberrant immune response as a causative mechanism in neurologic diseases has been extensively studied, is known as experimental autoimmune encephalomyelitis (EAE). While it reproduces essential aspects of the pathogenes

先行

FOIL

Michael P. Seed,Derek Gilroy,Mark Paul-Clark,Paul R. Colville-Nash,Dean Willis,Annette Tomlinson,Der