Titlebook: Immunosuppressant Analogs in Neuroprotection; Cesario V. Borlongan,Ole Isacson,Paul R. Sanberg Book 2003 Springer Science+Business Media N

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Blockade of Late-Onset Reduction of Muscarinic Acetylcholine Receptors by Immunosuppressants in Foreerve endings may be transiently or persistently destroyed. Although the molecular basis for the permanent neuronal destruction induced by transient ischemia has not been fully determined, there are several plausible hypotheses. Ischemic neuronal damage may be induced by several factors, including the following mechanisms:

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Introduction: Immunosuppressants as Neuroprotective Agentsimpact on medicine in their new role as neuroprotectants. The elucidation of the mechanisms of action of these drugs in ischemia, trauma, and neurodegeneration has enhanced our understanding of some of the complexities of neuronal death and survival.

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Radiation