Titlebook: Immunopathogenetic Mechanisms of Arthritis; J. A. Goodacre,W. Carson Dick Book 1988 MTP Press Limited 1988 genetics.immunity.infection.inf

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Dendritic cells in inflammatory joint disease,ence of cytokines such as interleukin-1 (IL-1). or from the release of degradative enzymes from macrophages. but, whatever the final pathway for destruction of the joint, the underlying activation appears to be immunological. This chapter is not mainly concerned with the afferent or effector mechani

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T-cell activation and function,d modify the immune response. Major recent advances regarding T-cell function and differentiation have led to the definition of unique sub-populations of T lymphocytes by characteristic cell surface glycoproteins (Table 5.1).. Probably the most important breakthrough has resulted in major understand

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Immune reactions in the rheumatoid synovial tissue,ch regulatory and effector mechanisms are involved in the formation of the rheumatic lesions and, most importantly, (ii) what is the specificity (specificities) of the immune reactions responsible for the subsequent development of the multiple immunological and inflammatory events that can be observ

Meander

Experimental animal models of chronic arthritis,mmunological reaction directed at either: (i) an exogenous agent that localizes to the joints or (ii) an articular autoantigen. These concepts, as suggested by studies of arthritis in animals, will almost certainly be modified in the future since immunological reactions directed at exogenous or host

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摘要

locus-ceruleus