Titlebook: Immunological Aspects of Liver Disease; Howard C. Thomas,Peter A. Miescher,Hans J. Mueller Book 1982 Springer-Verlag Berlin Heidelberg 198

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Book 1982to "self‘ components. Autoimmune disease occurs as a failure ofthis system either as a result of a primary defect in the regulatory apparatus (primary autoimmunization) or because of a change in the antigenicity of the tissues (secondary autoimmunization). Autoaggressive reactions are characterised

recession

Drug-Induced Chronic Hepatitis,. All individuals are susceptible, the lesion is reproducible in animals and there is a direct dose relationship. Even after severe hepatic necrosis, should the patient survive, agents producing a direct hepatotoxic reaction seldom lead to a chronic lesion.

enormous

一再困擾

Genetic Determinants of Autoimmune Chronic Active Hepatitis,lity antigens, of relevance to tissue transplantation, and associations between certain immunopathic diseases and HLA specificities [26, 31]. However, despite much experimental work and theoretical speculation, no unifying concept relating MHC-linked Ir genes effects and MHC- linked disease susceptibility has yet emerged.

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大量

Immunopathogenesis of the Extrahepatic Manifestations of Hepatitis B Virus Infection,ese syndromes and their comparison with animal models of immune complex injury, we have made progress in understanding the pathogenesis of these extrahepatic features; however, many important questions about the association among HBV, immune complexes, and extrahepatic tissue injury remain unanswered.

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Pathogenic Mechanisms in Primary Biliary Cirrhosis,moral immunity. Patients exhibit a generalised state of immune hyporesponsiveness being anergic to DNCB [15] and producing a diminished antibody response to parenteral antigenic challenge [16, 57]. They also exhibit failure of the immune response to convert from IgM to IgG antibody synthesis [57].