Titlebook: Idiopathic Dilated Cardiomyopathy; Cellular and Molecul Hans-Reiner Figulla,Reinhard Kandolf,Bruce McManus Conference proceedings 1993 Spri

多嘴多舌

假裝是你

分散

牽連

和音

The Promoter of the Human Cardiac ,-Myosin Heavy-Chain Gene Is Controlled by an Upstream Enhancer The two cardiac genes are located on chromosome 14; the skeletal muscle genes are on chromosome 17 [6, 14, 15]. The cardiac .-MHC is fast with respect to ATPase activity and speed of contraction; in contrast, .-MHC is slow. The two isoforms are distinctively distributed within the heart: . is found

無(wú)可非議

Growth Factor Expression in the Cardiac Myocyte or as a result of congenital heart disease in both the young and the adult mammal may result from cellular and molecular mechanisms which have common features. Protein growth factors are known to play a critical role in the cycle of normal cell division, differentiation and growth of cells from a v

偉大

Recessive

Detection of Disruption of Sarcolemmal Membrane by Indium-111 Labeled Antimyosin Antibody Scan presence or absence of myocardial cell necrosis with antimyosin scan. To our astonishment, we discovered that 50%-60% of patients with idiopathic dilated cardiomyopathy showed acute myocardial cell necrosis based on antimyosin scan results [1, 2]. Patients with myocarditis may present with congesti

ILEUM

installment