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Titlebook: Hypoxia; Through the Lifecycl Robert C. Roach,Peter D. Wagner,Peter H. Hackett ( Conference proceedings 2003 The Editor(s) (if applicable)

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發(fā)表于 2025-3-21 18:55:29 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Hypoxia
副標題Through the Lifecycl
編輯Robert C. Roach,Peter D. Wagner,Peter H. Hackett (
視頻videohttp://file.papertrans.cn/431/430804/430804.mp4
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Hypoxia; Through the Lifecycl Robert C. Roach,Peter D. Wagner,Peter H. Hackett ( Conference proceedings 2003 The Editor(s) (if applicable)
描述The International Hypoxia Symposium convenes biannually to bring together international experts from many fields to explore the state of the art in normal and pathophysiological responses to hypoxia. Representatives from five continents and 32 countries joined together in February 2003 for four days in the dramatic mountains of Banff, Alberta. As editors of the Proceedings of the International Hypoxia Symposia, we strive to maintain a 26 six year tradition of presenting a stimulating blend of clinical and basic science papers focused on hypoxia. Topics covered in 2003 include hibernation and hypoxia, hypoxia and fetal development and new advances in high altitude pathophysiology, oxidative stress and membrane damage, hypoxic regulation of blood flow, heat shock proteins in hypoxia, and future directions in hypoxia research. In 2003 we also had the privilege ofhonoring John W. Severinghaus as a friend, colleague, mentor and inspiration to many in the field. Tom Hornbein‘s personal tribute to John Severinghaus is the first chapter in this volume, followed by an entertaining update of the history of the discovery of oxygen written by John Severinghaus.
出版日期Conference proceedings 2003
關鍵詞Flow; hypertension; pathophysiology; physiology; skeletal muscle
版次1
doihttps://doi.org/10.1007/978-1-4419-8997-0
isbn_softcover978-1-4613-4753-8
isbn_ebook978-1-4419-8997-0Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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發(fā)表于 2025-3-21 23:59:51 | 只看該作者
Fire-Air and Dephlogistication,a new element, oxygen, which combined with hydrogen to make water. He disproved the phlogiston theory but Priestley called it dephlogisticated air until his death 30 years later. Scandanavians learn that a Swedish apothecary Carl Wilhelm Scheele beat Priestley by 2 years but was deprived of credit b
板凳
發(fā)表于 2025-3-22 04:00:54 | 只看該作者
Mammalian Hibernation,ure falls to 0–5??C, and physiological functions are strongly suppressed. Hibernation is a closely regulated process that includes multiple controls on gene transcription and protein translation, the primary subjects of this review. Recent studies by our lab and others have used multiple techniques
地板
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Current Paradigms in Cellular Oxygen Sensing,ry, and the resistance of cells against the consequences of severe hypoxia. These responses are required for embryonic development and for survival through adulthood. Although much has been learned about the signaling pathways that are activated in hypoxic cells, the underlying mechanism of O.sensin
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發(fā)表于 2025-3-22 14:08:17 | 只看該作者
Why is Erythropoietin Made in the Kidney?, the production of erythropoietin,which further drives the production of red blood cells in the bone marrow,explains how the hematocrit is . it does not speak to how the hematocrit is . The regulation of the hematocrit requires the coordination of the plasma volume and the red cell mass. By controll
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發(fā)表于 2025-3-22 17:35:17 | 只看該作者
Hypoxia and High Altitude,gen partial pressure (pO.). Intensive research during the last years revealed that the molecular mechanism behind the regulation of erythropoietin is ubiquitous and has far more implications than first thought. Erythropoietin regulation results from the activation of the hypoxia-inducible factor-1 (
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Hypoxia and Rho/Rho-Kinase Signaling,tions, including cell contraction, migration, gene expression, proliferation, and differentiation. Rho/Rho-kinase signaling has an obligatory role in embryonic cardiac development, and low-level chemical activation of Rho promotes branching morphogenesis in fetal lung explants. Gebb has found that h
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