Titlebook: Hypertrophic Cardiomyopathy; The Therapeutic Role M. Kaltenbach,S. E. Epstein Conference proceedings 1982 Springer-Verlag Berlin Heidelberg

錯(cuò)誤

Synopsisl and other calcium antagonist drugs, as well as into the pathophysiologic mechanisms that may contribute importantly to the natural history of the disease. Thus, it is clear that verapamil diminishes the left ventricular outflow tract gradient present in those patients with the obstructive form of

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muffler

Hemodynamics and Contractility After Oral, Intravenous, and Intracoronary Application of Calcium Ants study was designed to verify a possible negative inotropic effect of both drugs. Isovolumic and auxotonic contractility were measured following oral application, intravenous infusion, intravenous bolus injection, and intracoronary administration..Given orally, . has no cardiodepressive effect. Str

Dysarthria

Effect of Verapamil on Left Ventricular Isovolumic Relaxation Time and Regional Left Ventricular Fil and a disturbed left ventricular filling pattern [1–5]. Recent experimental studies revealed that calcium antagonists may improve impaired left ventricular relaxation caused by ischemia or hypoxia [6, 7]. Based on these experimental results, it was the purpose of the present study to examine whethe

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Offensive

Weimarer Nietzsche-Bibliographie in 5 B?ndenrophic cardiomyopathy. Most commonly (52% of 125 patients) hypertrophy involved substantial portions of both the ventricular septum and anterolateral left ventricular free wall (type III). In other patients, hypertrophy was confined to the anterior portion of ventricular septum (type I), involved th

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https://doi.org/10.1007/978-3-476-02730-6th hypertrophic cardiomyopathy. Cellular disorganization was both common and extensive (mean area of tissue section disorganized 35 ± 4%) in the ventricular septum and proved to be a highly specific and sensitive marker for hypertrophic cardiomyopathy. Disorganization was also substantial (24 ±3%) i

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Weimarer Nietzsche-Bibliographie in 5 B?nden and left ventricle obtained during catheterization [1–3]. Characteristic changes in myocardial architecture, predominantly of the septal region, have been described by many authors [1, 2, 4, 5]. The reliability of endomyocardial biopsy in diagnosing hypertrophic cardiomyopathy remains controversial

discord

Weimarer Nietzsche-Bibliographie in 5 B?ndenaneously occurring or experimentally produced animal model of this disease suitable for controlled experimentation would be potentially useful. The three papers presented in this chapter describe some of the current data concerning available animal models and their potential use in experimental stud