Titlebook: Hypertension and the Heart; Alberto Zanchetti,Richard B. Devereux,Sergio Gorin Book 1997 The Editor(s) (if applicable) and The Author(s),

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Hypertension Differentially Affects the Expression of the Gap Junction Protein Connexin43 in Cardiacels aggregated at gap junctions. Connexin43 (Cx43) is one of the predominant membrane proteins forming junctional channels in the cardiovascular system. This study was undertaken to assess its expression during experimental hypertension. Rats were made hypertensive by clipping one renal artery (two-

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Modulation of Cardiac Hypertrophy by Estrogensle to investigate the molecular mechanisms..Most biochemical work in the last 50 years has focused on the characterization of the steroid hormones involved in gender specificity. More recently, the cloning of the steroid receptors and characterization of the signaling pathways through these proteins

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Salt Sensitivity and Left Ventricular Hypertrophyn (EH).. The Framingham study. demonstrated that in 75% of all patients in whom heart failure developed, EH was the underlying disease. Moreover, EH accelerates coronary arteriosclerosis leading to myocardial ischemia in patients with genetic predisposition., a phenomenon that exacerbates in the pre

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The Renin-Angiotensin System Gene Polymorphism and Left Ventricular Hypertrophylearly demonstrated in the Framingham Study. On the other hand, inhibitors of angiotensin-converting enzyme (ACE) have been shown to have a beneficial effect on LVH. In the SOLVD trial, treatment by ACE-inhibitors was associated with a reduction of the incidence of myocardial infarction (MI) and uns

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Renin-Angiotensin System Gene Polymorphisms and Left Ventricular Hypertrophyin-converting enzyme (ACE), and cardiovascular disease. However, there is lack of agreement that the ACE polymorphism is associated with left ventricular hypertrophy (LVH) in hypertension. A possible paradigm for the development of LVH involves the ACE gene polymorphism influencing cardiac mass by a

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