Titlebook: Host-Parasite Cellular and Molecular Interactions in Protozoal Infections; K.-P. Chang,David Snary Conference proceedings 1987 Springer-Ve

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書目名稱Host-Parasite Cellular and Molecular Interactions in Protozoal Infections影響因子(影響力)學(xué)科排名




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Palpitation

milligram

Nonporous

使增至最大

1010-8793 nsiderable morbidity and mortality, especially in the third world countries. These diseases are caused by a group of protozoa which have, over the years, undergone evolutionary adaptation to live often intracellularly in a parasitic way of life. So well-adapted have they become that they recognize t

cliche

Evolution of Antigenic Variation in African Trypanosomes the parasites while in the bloodstream of the host. On any one trypanosome, at any one time, this coat is essentially homogeneous, being comprised of a single glycoprotein species, known as the variant surface glycoprotein or VSG (Cross, 1975; Vickerman and Luckins, 1969).

自愛

Metabolism of Trypanothione and Glutathionylspermidine in Trypanosomatidsein synthesis and regulation of enzyme activity C3]. The intracellular ratio of reduced to oxidized glutathione (GSH : GSSG) is kept high (GSSG < 1% of the total) due to the presence of the NADPH-dependent enzyme, glutathione reductase [E.C. 1.6.4.2].

delta-waves

一起平行

The Promastigote Surface Protease of Leishmania three strains of Leishmania previously shown by surface radioiodination, peptide mapping and antigenic crossreactivity to express gp63: L. major LRC-L137, L. donovani infantum LEM 75, and L. tropica LRC-L32 (Etges et al. 1985).

rectocele

A Specific Inhibitor of Trypanosoma Cruzi Neuraminidase Found in Human Plasmablished between 1903 and 1935 he described the disease, its etiology, its vectors and its reservoirs. Chagas’ disease predominantly striking the rural population, still represents an exciting challenge to the scientific community because the molecular mechanisms of its pathogenicity have yet to be unravelled.