Titlebook: Hormone Resistance Syndromes; J. Larry Jameson Book 1999 Springer Science+Business Media New York 1999 ACTH.Diabetes.Diabetes mellitus.glu

MEDAL

書目名稱Hormone Resistance Syndromes影響因子(影響力)




書目名稱Hormone Resistance Syndromes影響因子(影響力)學(xué)科排名




書目名稱Hormone Resistance Syndromes網(wǎng)絡(luò)公開度




書目名稱Hormone Resistance Syndromes網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Hormone Resistance Syndromes被引頻次




書目名稱Hormone Resistance Syndromes被引頻次學(xué)科排名




書目名稱Hormone Resistance Syndromes年度引用




書目名稱Hormone Resistance Syndromes年度引用學(xué)科排名




書目名稱Hormone Resistance Syndromes讀者反饋




書目名稱Hormone Resistance Syndromes讀者反饋學(xué)科排名

legislate

Pseudohypoparathyroidism,These clinical and bio-chemical features were consistent with the diagnosis of hypoparathyroidism. However, unlike patients with parathyroid hormone (PTH) deficiency, these patients failed to show a rise in serum calcium or urinary phosphate in response to injected parathyroid extract (.). Albright

Mast-Cell

Hereditary Resistance to Vitamin D,ctive form of vita-min D, 1,25-dihydroxyvitamin D [1,25(OH).D], binds with high affinity to specific vitamin D receptors (VDR) located in the nucleus of target cells. The VDR is a member of the steroid-thyroid-retinoid receptor gene superfamily of nuclear transcription factors that mediate the biolo

mydriatic

Arboreal

inhumane

大吃大喝

Diabetes Mellitus,es in the metabolism of glucose, fat, and proteins .. For example, when insulin secretion is impaired by destruction of the pancreatic 13-cell (e.g., autoimmune destruction of the (3-cell in type 1 diabetes mellitus), this leads to characteristic metabolic derangements, including hyperglycemia and k

徹底明白

LH Insensitivity Syndrome,is syndrome are described. First, an introduction is given of the physiological functions of LH and human chorion gonadotropin (hCG) during prenatal sex differentiation, puberty, and in the adult state. Subsequently, LH insensitivity syndrome is described in detail in males and females as well as se

fetter

FSH Resistance,rian and testicular function is well known. As a consequence, the ability of gonadotropins to stimulate ovarian follicular growth has not only been scientifically studied, but also vigorously exploited in the treatment of infertile women, although the exact molecular mechanisms of gonadotropin actio

interference