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Titlebook: Hormonal Control of Cell Cycle; Shlomo Melmed,Henri Rochefort,Yves Christen Conference proceedings 2008 Springer-Verlag Berlin Heidelberg

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發(fā)表于 2025-3-21 19:37:04 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Hormonal Control of Cell Cycle
編輯Shlomo Melmed,Henri Rochefort,Yves Christen
視頻videohttp://file.papertrans.cn/429/428281/428281.mp4
概述Includes supplementary material:
叢書名稱Research and Perspectives in Endocrine Interactions
圖書封面Titlebook: Hormonal Control of Cell Cycle;  Shlomo Melmed,Henri Rochefort,Yves Christen Conference proceedings 2008 Springer-Verlag Berlin Heidelberg
出版日期Conference proceedings 2008
關(guān)鍵詞Cyclin E; breast cancer; estrogens; growth factor; hormone signaling; mitosis; nuclear receptors; prostrate
版次1
doihttps://doi.org/10.1007/978-3-540-73855-8
isbn_softcover978-3-642-09310-4
isbn_ebook978-3-540-73855-8Series ISSN 1861-2253 Series E-ISSN 1863-0685
issn_series 1861-2253
copyrightSpringer-Verlag Berlin Heidelberg 2008
The information of publication is updating

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1861-2253 Overview: Includes supplementary material: 978-3-642-09310-4978-3-540-73855-8Series ISSN 1861-2253 Series E-ISSN 1863-0685
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https://doi.org/10.1007/978-3-540-73855-8Cyclin E; breast cancer; estrogens; growth factor; hormone signaling; mitosis; nuclear receptors; prostrate
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DNA Replication Origins, Development, and Cancer, and its transmission to the offspring. In simple organisms, such as the bacteria ., a single DNA replication origin is used, fromwhich replication forksmigrate in opposite directions to allowthe duplication of the entire genome. In complex eukaryotes, such as multicellular organisms,multiple DNArep
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Getting In and Out of Mitosis, genome, and how to prevent cell separation before chromosome segregation. Both these problems are solved by controlling when two specific proteins are destroyed: securin, an inhibitor of chromosome segregation, and cyclin B, which inhibits cell separation (cytokinesis). It has recently become clear
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A Novel Function for Cyclin E in Cell Cycle Progression,tant can reconstitute cyclin E’s function in cyclin E-null cells. Kinase-deficient cyclin E1 is loaded onto chromatin during G0 → S progression, it restores MCM incorporation and it facilitates S phase entry of cyclin E-null cells. We also observed that, in wild-type cells, cyclin E is loaded onto D
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