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Titlebook: Hormonal Carcinogenesis; Proceedings of the F Jonathan J. Li,Satyabrata Nandi,Sara Antonia Li Conference proceedings 1992 Springer-Verlag N

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發(fā)表于 2025-3-21 19:14:47 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Hormonal Carcinogenesis
副標(biāo)題Proceedings of the F
編輯Jonathan J. Li,Satyabrata Nandi,Sara Antonia Li
視頻videohttp://file.papertrans.cn/429/428276/428276.mp4
圖書封面Titlebook: Hormonal Carcinogenesis; Proceedings of the F Jonathan J. Li,Satyabrata Nandi,Sara Antonia Li Conference proceedings 1992 Springer-Verlag N
描述In the past decade there has been a growing public interest and resurgence in research in the field of hormonal carcinogenesis. This is due to the widespread use of therapeutic hormonal agents worldwide and to the increasing awareness of the causal association of hormones, both endogenous and exogenously administered, and a variety of human cancers. These associations include estrogens in uterine, cervical, vaginal, liver, testicular, prostatic, and possible breast cancers; progesterone and progestational hormones in breast cancer; androgens and anabolic steroids in hepatic and prostatic cancers. Additionally, gonadotrophins playa role in the etiology of ovarian and testicular cancers and thyroid-stimulating hormones in thyroid cancers. Therefore, hormonal carcinogenesis encompasses the study of both natural and synthetic hormonal agents, including growth factors and other peptide and protein factors, which contribute substantially to the etiology of both human and animal neoplasms, benign or malignant. Hormones may be involved in all aspects of neoplastic transformation, including initiation, promotion, and progression, and the inhibition of these processes. There are a number of
出版日期Conference proceedings 1992
關(guān)鍵詞breast cancer; carcinogenesis; hormones; liver; steroids
版次1
doihttps://doi.org/10.1007/978-1-4613-9208-8
isbn_softcover978-1-4613-9210-1
isbn_ebook978-1-4613-9208-8
copyrightSpringer-Verlag New York, Inc. 1992
The information of publication is updating

書目名稱Hormonal Carcinogenesis影響因子(影響力)




書目名稱Hormonal Carcinogenesis影響因子(影響力)學(xué)科排名




書目名稱Hormonal Carcinogenesis網(wǎng)絡(luò)公開度




書目名稱Hormonal Carcinogenesis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Hormonal Carcinogenesis被引頻次




書目名稱Hormonal Carcinogenesis被引頻次學(xué)科排名




書目名稱Hormonal Carcinogenesis年度引用




書目名稱Hormonal Carcinogenesis年度引用學(xué)科排名




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Introductions. Most of these crystals were apparently sex hormones whose chemical structures were determined during the 1920s to 1930s. By the end of the 1930s, pure androgenic steroids were used in the treatment of some patients, and Huggins demonstrated that metastatic prostatic cancer could be treated by castration (1).
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Oxidation of Estrogens and Other Steroids by Cytochrome P-450 Enzymes: Relevance to Tumorigenesis cell proliferation. Similarly, higher estrogen and progesterone levels increase breast cancer risk. However, the use of the artificial steroids in oral contraceptives can reduce endometrial cancer risk (1). The androgen testosterone may be a factor in prostatic cancer.
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The Conjoint Actions of Androgens and Estrogens in the Induction of Proliferative Lesions in the Ratanging levels of circulating androgens and/or estrogens or an altered ratio of the two classes of steroids in plasma are important determinants of prostatic carcinogenesis in the human male. Moreover, the influence of sex hormones could begin in the early decades of a man’s life and have a long-lasting impact on the prostate.
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Introductionr. With the demonstration that only the transformed receptor has the ability to influence RNA synthesis in isolated target cell nuclei, there emerged a general concept of steroid hormone action, in which the role of the steroid is to convert the native receptor protein to a modulator of transcription (3).
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Conference proceedings 1992espread use of therapeutic hormonal agents worldwide and to the increasing awareness of the causal association of hormones, both endogenous and exogenously administered, and a variety of human cancers. These associations include estrogens in uterine, cervical, vaginal, liver, testicular, prostatic,
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