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Titlebook: Histophilus somni; Biology, Molecular B Thomas J. Inzana Book 2016 The Editor(s) (if applicable) and The Author(s), under exclusive license

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書(shū)目名稱Histophilus somni
副標(biāo)題Biology, Molecular B
編輯Thomas J. Inzana
視頻videohttp://file.papertrans.cn/428/427325/427325.mp4
概述Provides an overall review of the bacterium‘s genetics and what DNA- and RNA-sequencing can tell us about the organism‘s physiology.Reviews the interaction of H. somni with host endothelials cells and
叢書(shū)名稱Current Topics in Microbiology and Immunology
圖書(shū)封面Titlebook: Histophilus somni; Biology, Molecular B Thomas J. Inzana Book 2016 The Editor(s) (if applicable) and The Author(s), under exclusive license
描述.This volume reviews the current understanding of the taxonomy, disease syndromes, genetics, biology, and pathogenic factors of .Histophilus somni., as well as the host immune response to this pathogen. .H. somni. is one of the most important bacterial pathogens in cattle and other ruminants, and its virulence factors are highly conserved with .Haemophilus influenzae .and other members of the .Pasteurellaceae.. .H. somni. has been recognized as a major cause of thrombotic meningoencephalitis, respiratory disease syndromes, myocarditis, reproductive disease syndromes, polyarthritis, mastitis, ocular disease, and septicemia. The only known habitats of .H. somni .are the mucosal surfaces of ruminants, making this bacterium an opportunistic pathogen. Although it is capable of causing inflammation at systemic sites and is toxic to epithelial and phagocytic cells, the bacterium’s wide array of virulence factors act primarily as a defense against, or to escaperecognition from, host innate and adaptive immunity..
出版日期Book 2016
關(guān)鍵詞Pasteurellaceae; Bacterial pathogen; Biofilm formation in host colonization; Feedlot cattle diseases; Ho
版次1
doihttps://doi.org/10.1007/978-3-319-29556-5
isbn_softcover978-3-319-80598-6
isbn_ebook978-3-319-29556-5Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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Interactions of , with Host Cells,which also can enhance tissue factor activity on the endothelium surface. When exposed to ., bovine neutrophils and mononuclear phagocytes form extracellular traps in vitro. Ongoing research is investigating how the interplay among endothelial cells, platelets, and leukocytes might contribute to the
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