Titlebook: Hepatic Stellate Cells; Methods and Protocol Ralf Weiskirchen,Scott L. Friedman Book 2023 The Editor(s) (if applicable) and The Author(s),

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書目名稱Hepatic Stellate Cells影響因子(影響力)




書目名稱Hepatic Stellate Cells影響因子(影響力)學(xué)科排名




書目名稱Hepatic Stellate Cells網(wǎng)絡(luò)公開度




書目名稱Hepatic Stellate Cells網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Hepatic Stellate Cells被引頻次




書目名稱Hepatic Stellate Cells被引頻次學(xué)科排名




書目名稱Hepatic Stellate Cells年度引用




書目名稱Hepatic Stellate Cells年度引用學(xué)科排名




書目名稱Hepatic Stellate Cells讀者反饋




書目名稱Hepatic Stellate Cells讀者反饋學(xué)科排名

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Testing Cell Migration, Invasion, Proliferation, and Apoptosis in Hepatic Stellate Cells, cells (SECs), and hepatic stellate cells (HSCs). Normally, in their quiescent state, HSCs are a reservoir for vitamin A, but in response to hepatic injury, they become activated myofibroblasts that play a key role in the hepatic fibrotic response. Activated HSCs express extracellular matrix (ECM) p

Aggressive

Phalloidin Staining for F-Actin in Hepatic Stellate Cells, contractile, α-smooth muscle actin-positive myofibroblast. These cells acquire properties that are strongly associated with the reorganization of the actin cytoskeleton. Actin possesses a unique ability to polymerize into filamentous actin (F-actin) form its monomeric globular state (G-actin). F-ac

Foreshadow

Retinyl Ester Analysis by Orbitrap Mass Spectrometry, of retinyl ester species by high-resolution mass spectrometry. Retinyl esters are extracted by the method of Bligh and Dyer and subsequently separated by HPLC in runs of 40?min. The retinyl esters are identified and quantified by mass spectrometry analysis. This procedure enables the highly sensiti

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rectum

Isolation of Hepatic Stellate Cells and Lymphocytes for Co-culture Systems,unt of extracellular matrix such as collagen fibers. However, recent lines of evidence have also highlighted the immunoregulatory functions of HSCs, in which they interact with diverse hepatic lymphocytes to produce cytokines and chemokines, release extracellular vesicles, or express specific ligand

Guileless

Working with Immortalized Hepatic Stellate Cell Lines,ved considerable attention in studies investigating fundamental features of hepatic fibrosis. However, the limited supply and ever-increasing demand for these cells, combined with the additional tightening of formal standards in animal welfare policy, make working with these primary cells increasing

樂章

Induction of Obstructive Cholestasis in Mice,eriportal biliary fibrosis. These changes are time-dependent and based on excess accumulation of bile acids in the liver. This in turn causes damage of hepatocytes and functional loss, leading to recruitment of inflammatory cells. Liver resident pro-fibrogenic cells facilitate extracellular matrix s

commonsense

Mouse Models for Hepatic Stellate Cell Activation and Liver Fibrosis Initiation, not available. Accordingly, there is a strong need to conduct intensive basic research, which also includes the necessity to use animal models to evaluate new anti-fibrotic therapy concepts. Numerous mouse models of liver fibrogenesis have been described. This involves chemical, nutritional, surgic