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Titlebook: Hepatic Encephalopathy; Kevin D. Mullen,Ravi K. Prakash Book 2012 The Editor(s) (if applicable) and The Author(s), under exclusive license

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樓主: Hermit
41#
發(fā)表于 2025-3-28 15:05:00 | 只看該作者
ically vocal Twitter users, most events during the campaign did not, and left, therefore, no visible traces in the dynamics of Twitter messages referring to politics. The same was true for the analysis of political controversies, where personalized scandalization and issues related to Internet regul
42#
發(fā)表于 2025-3-28 21:46:02 | 只看該作者
43#
發(fā)表于 2025-3-29 01:03:18 | 只看該作者
o present any results from the study, but to highlight a proposed methodology for capturing data which is applicable to an area of study which is often overlooked — that is, candidate-level online campaigning. This methodology aims to improve upon past studies into these areas with an updated approa
44#
發(fā)表于 2025-3-29 06:51:13 | 只看該作者
2197-7399 ue to gastroenterologists, hepatologists, pathologists, medical residents, fellows, internists and general practitioners who treat patients with hepatic encephalopathy..978-1-4939-0390-0978-1-61779-836-8Series ISSN 2197-7399 Series E-ISSN 2197-7704
45#
發(fā)表于 2025-3-29 07:30:38 | 只看該作者
46#
發(fā)表于 2025-3-29 14:41:54 | 只看該作者
Role of Ammonia in the Pathogenesis of Hepatic Encephalopathyrrelation between the advancement of HE symptoms and the increase in blood ammonia. The risk of development of HE cirrhotic patients is associated with increased ammonia production from glutamine in the intestines or kidney. Both clinical and animal model studies favor the role of ammonia in inducin
47#
發(fā)表于 2025-3-29 16:52:15 | 只看該作者
48#
發(fā)表于 2025-3-29 20:26:24 | 只看該作者
49#
發(fā)表于 2025-3-30 02:22:47 | 只看該作者
50#
發(fā)表于 2025-3-30 08:07:29 | 只看該作者
The Role of Natural Benzodiazepines Receptor Ligands in Hepatic Encephalopathyancing the action of GABA was originally suggested by anecdotal observations of ameliorations of HE following administration of a BZ receptor antagonist. The term natural BZs has been introduced for BZ receptor ligands which have been demonstrated in brain, cerebrospinal fluid, and plasma of normal
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