Titlebook: Hepatic Encephalopathy; Kevin D. Mullen,Ravi K. Prakash Book 2012 The Editor(s) (if applicable) and The Author(s), under exclusive license

HILAR

暫時別動

delegate

In this chapter, we concentrate on the structure of stories abandoning the context of scripts. We will return to the scripts and the graphs related to them, such as graph AB, in Chap.?.. We currently assume that the social network is given and is connected to the narrative by human intelligence and not computed using the AB graph.

秘密會議

Kevin D. Mullen MD, FRCPI,Ravi K. Prakash MBBS, MD, MRCP (UK)We now lay the foundation to model narrative and conflict in social and literature networks. This chapter demonstrates how to mechanically construct graphs from scripts. In doing so, we invite machines to participate in the reading process.

BLA

We continue our discussion of narrative conflict through the lens of war. We employ the metaphor of war as a Platonic ideal of conflict. In the previous chapter, we looked at direct attacks and modeled the network distances as metric space.

狂熱文化

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Clinical Gastroenterologyhttp://image.papertrans.cn/h/image/425689.jpg

RAG

Role of Ammonia in the Pathogenesis of Hepatic Encephalopathynce. The edema-inducing effect is to a considerable degree mediated by glutamine, which acts via two complementary mechanisms: (a) by increasing the intracellular osmotic pressure; (b) by causing mitochondrial damage following its entry to the inside of the organelle, where it unloads ammonia in a g

血統(tǒng)

Neuroinflammation in the Pathogenesis of Hepatic Encephalopathymechanisms between the failing liver and the brain leading to neuroinflammation is unknown, mechanisms involving blood–brain cytokine transfer and brain lactate have been proposed. Neuroinflammatory responses in liver failure result in upregulation of translocator protein (TLP), a mitochondrial memb

Isthmus

Inflammation and Hepatic Encephalopathybe implicated. Ammonia has also been shown to induce immune dysfunction with the release of reactive oxygen species from neutrophils which contribute to oxidative stress and systemic inflammation. Not only can this exacerbate the cerebral effects of ammonia but it may also make the individual vulner