Titlebook: Hepatic De Novo Lipogenesis and Regulation of Metabolism; James M. Ntambi Book 2016 Springer International Publishing Switzerland 2016 De

借喻

變白

HALO

Indebted

Molecular Mechanisms of Lipotoxicity in Nonalcoholic Fatty Liver Disease,), the more severe form of NAFLD, is associated with increased risk of cardiovascular morbidity and mortality, insulin resistance (IR), type 2 diabetes mellitus (T2DM), hepatic steatosis, elevated circulating levels of free fatty acids (FFAs) and hepatocyte apoptosis. Because lipid-induced toxicity,

forthy

勉勵(lì)

Constant

Lipid Droplet Proteins and Hepatic Lipid Metabolism, passive depot of excess lipids but is a metabolically active organelle that interacts with endoplasmic reticulum, mitochondria and other cellular compartment to regulate fate of intracellular lipids. As the increase in size and number of LD is a key feature of hepatosteatosis, information regarding

EWER

Influence of Gut Microbiota on Hepatic Lipogenesis and Disease Pathogenesis,y of biochemical pathways that are central in keeping energy homeostasis. Loss of this delicate balance, however, may lead to a spectrum of liver diseases such as NAFLD, NASH, fibrosis, cirrhosis and HCC. The etiology of liver disorders is often multifactorial and greatly influenced by host genetics

MIRTH

outset