Titlebook: Hemostatic Mechanisms and Metastasis; Kenneth V. Honn,Bonnie F. Sloane Book 1984 Martinus Nijhoff Publishing, Boston 1984 carcinoma.cell.c

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Fibrin Formation: Implications for Tumor Growth and Metastasis,selves, their abnormal morphology, their altered metabolism and more recently, their panoply of surface antigens. Yet, tumors are comprised not only of malignant cells but also of stroma. In the case of many solid tumors stroma may actually comprise the bulk of the mass perceived as tumor with the n

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Perspectives on the Role of Platelets in Hemostasis and Thrombosis, of hemostasis and in the pathogenesis of thrombosis. When platelets are stimulated, they release biologically active constituents which interact with vessel wall cells, plasma coagulation factors and circulating blood cells to modulate hemostasis. In this review we will discuss platelet adhesion, t

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Mechanisms of Platelet Aggregation by Murine Tumor Cell Sheddings, been fully elucidated preliminary evidence suggests that several mechanisms may exist. With human tumor cells platelet aggregation may be induced via mechanisms involving the leakage of tumor cell ADP (2, 4, 5; see also Chapters 11 and 12). In rodents aggregation may occur with both whole cells and

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Calcium Channel Blockers: Inhibitors of Tumor Cell-Platelet-Endothelial Cell Interactions,ger that modulates biochemical processes appropriate to the specific cell type. Calcium antagonists by preventing release of Ca. from intracellular stores, by peventing influx of external Ca. or by inhibiting the effects of Ca. through interactions with Ca.-binding proteins such as calmodulin can in

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